Silencing of BNIP3 results from promoter methylation by DNA methyltransferase 1 induced by the mitogen-activated protein kinase pathway. - Wikidata - wikimedia - TriplyDB

Silencing of BNIP3 results from promoter methylation by DNA methyltransferase 1 induced by the mitogen-activated protein kinase pathway.

Silencing of BNIP3 results from promoter methylation by DNA methyltransferase 1 induced by the mitogen-activated protein kinase pathway.

http://www.wikidata.org/entity/Q37461041

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KRAS Genomic Status Predicts the Sensitivity of Ovarian Cancer Cells to Decitabine.Increased NQO1 but not c-MET and survivin expression in non-small cell lung carcinoma with KRAS mutations Hedgehog signaling pathway regulates autophagy in human hepatocellular carcinoma cells.Molecular differences between cerebral blood volume and vessel size in glioblastoma multiforme.Structure, function, and epigenetic regulation of BNIP3: a pathophysiological relevance.Methylation-mediated silencing of miR-124 facilitates chondrogenesis by targeting NFATc1 under hypoxic conditions.Methylation of BNIP3 in pancreatic cancer inhibits the induction of mitochondrial-mediated tumor cell apoptosis.125I seed irradiation induces up-regulation of the genes associated with apoptosis and cell cycle arrest and inhibits growth of gastric cancer xenografts.BNIP3 induces apoptosis and protective autophagy under hypoxia in esophageal squamous cell carcinoma cell lines: BNIP3 regulates cell death.DNA methylation of the promoter region of bnip3 and bnip3l genes induced by metabolic programming

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Silencing of BNIP3 results from promoter methylation by DNA methyltransferase 1 induced by the mitogen-activated protein kinase pathway.

http://www.wikidata.org/entity/Q37461041

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 May 2011

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Silencing of BNIP3 results fro ...... ivated protein kinase pathway.

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Silencing of BNIP3 results fro ...... ivated protein kinase pathway.

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type

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Silencing of BNIP3 results fro ...... ivated protein kinase pathway.

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Silencing of BNIP3 results fro ...... ivated protein kinase pathway.

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Silencing of BNIP3 results fro ...... ivated protein kinase pathway.

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Silencing of BNIP3 results fro ...... ivated protein kinase pathway.

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S10059-011-0065-Z

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Molecules and Cells

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Silencing of BNIP3 results fro ...... ivated protein kinase pathway.

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Hayyoung Lee

http://www.w3.org/2001/XMLSchema#string

Hyun-Jung An

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Sang-Gi Paik

http://www.w3.org/2001/XMLSchema#string

P2860

BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore

The E1B 19K/Bcl-2-binding protein Nip3 is a dimeric mitochondrial protein that activates apoptosis

Adenovirus E1B 19 kDa and Bcl-2 proteins interact with a common set of cellular proteins

DNMT1 and DNMT3b cooperate to silence genes in human cancer cells

Activation of Ras up-regulates pro-apoptotic BNIP3 in nitric oxide-induced cell death

Loss of BNIP3 expression is a late event in pancreatic cancer contributing to chemoresistance and worsened prognosis.

Silencing of the hypoxia-inducible cell death protein BNIP3 in pancreatic cancer.

BNIP3 expression is linked with hypoxia-regulated protein expression and with poor prognosis in non-small cell lung cancer.

Adenovirus E1B-19K/BCL-2 interacting protein BNIP3 contains a BH3 domain and a mitochondrial targeting sequence.

Silencing of the metastasis suppressor RECK by RAS oncogene is mediated by DNA methyltransferase 3b-induced promoter methylation.

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s10059-011-0065-z

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579-583

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scholarly article

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10.1007/S10059-011-0065-Z

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6

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2011-05-11T00:00:00Z

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