B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence. - Wikidata - wikimedia - TriplyDB

B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

http://www.wikidata.org/entity/Q37461823

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PD-L1 expression in human cancers and its association with clinical outcomes Tumor-targeted and immune-targeted monoclonal antibodies: Going from passive to active immunotherapy.PD1 is highly expressed in diffuse large B-cell lymphoma with hepatitis B virus infection.Cyclooxygenase-2 and B-cell lymphoma-2 expression in cystitis glandularis and primary vesicle adenocarcinoma.Biomarkers for Wilms Tumor: A Systematic Review Expression of programmed cell death ligand 1 is associated with poor overall survival in patients with diffuse large B-cell lymphoma.Anti-Programmed Cell Death (PD)-1 Immunotherapy for Malignant Tumor: A Systematic Review and Meta-Analysis.PI(3) kinase is associated with a mechanism of immunoresistance in breast and prostate cancer The complex role of B7 molecules in tumor immunology Co-expression of PD-L1 and p-AKT is associated with poor prognosis in diffuse large B-cell lymphoma via PD-1/PD-L1 axis activating intracellular AKT/mTOR pathway in tumor cells The role of programmed cell death-1 (PD-1) and its ligands in pediatric cancer.The PD-1/PD-L1 pathway: biological background and clinical relevance of an emerging treatment target in immunotherapy.Limitations and opportunities for immune checkpoint inhibitors in pediatric malignancies.Patterns of PD-1, PD-L1, and PD-L2 expression in pediatric solid tumors.Checkpoint Proteins in Pediatric Brain and Extracranial Solid Tumors: Opportunities for Immunotherapy.Are tumor-infiltrating lymphocytes protagonists or background actors in patient selection for cancer immunotherapy?Low Frequency of Programmed Death Ligand 1 Expression in Pediatric Cancers.Checkpoint inhibition in pediatric hematologic malignancies.Assessment of programmed death-ligand 1 expression and tumor-associated immune cells in pediatric cancer tissues.The dawn of "immune-revolution" in children: early experiences with checkpoint inhibitors in childhood malignancies.Differentiated thyroid carcinomas and their B7H1 shield.

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B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

http://www.wikidata.org/entity/Q37461823

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 20 March 2008

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

@en

B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

@nl

type

label

B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

@en

B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

@nl

prefLabel

B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

@en

B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

@nl

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J.JURO.2008.01.056

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The Journal of Urology

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B7-H1 expression in Wilms tumor: correlation with tumor biology and disease recurrence.

@en

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Christine M Lohse

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Douglas A Husmann

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Eugene D Kwon

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Jonathan C Routh

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Richard A Ashley

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Stephen A Kramer

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Thomas J Sebo

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P2860

B7-H1, a third member of the B7 family, co-stimulates T-cell proliferation and interleukin-10 secretion

Costimulatory B7-H1 in renal cell carcinoma patients: Indicator of tumor aggressiveness and potential therapeutic target

Tumor-associated B7-H1 promotes T-cell apoptosis: a potential mechanism of immune evasion

End stage renal disease in patients with Wilms tumor: results from the National Wilms Tumor Study Group and the United States Renal Data System.

Congestive heart failure after treatment for Wilms' tumor: a report from the National Wilms' Tumor Study group.

Treatment with nephrectomy only for small, stage I/favorable histology Wilms' tumor: a report from the National Wilms' Tumor Study Group.

Blockade of B7-H1 improves myeloid dendritic cell-mediated antitumor immunity.

Blockade of B7-H1 and PD-1 by monoclonal antibodies potentiates cancer therapeutic immunity.

Controversies and advances in the management of Wilms' tumour.

B7-H1 pathway and its role in the evasion of tumor immunity.

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1954-9; discussion 1959-60

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10.1016/J.JURO.2008.01.056

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2008-03-20T00:00:00Z

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