Induction of Th17 differentiation by corneal epithelial-derived cytokines - Wikidata - wikimedia - TriplyDB

Induction of Th17 differentiation by corneal epithelial-derived cytokines

Induction of Th17 differentiation by corneal epithelial-derived cytokines

http://www.wikidata.org/entity/Q37462911

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sPLA2-IIa amplifies ocular surface inflammation in the experimental dry eye (DE) BALB/c mouse model Prioritizing biological pathways by recognizing context in time-series gene expression data Phage display against corneal epithelial cells produced bioactive peptides that inhibit Aspergillus adhesion to the corneas Strain-dependent production of interleukin-17/interferon-γ and matrix remodeling-associated genes in experimental Candida albicans keratitis.The core mechanism of dry eye disease is inflammation.Dry eye as a mucosal autoimmune disease.Chemokine receptors CCR6 and CXCR3 are necessary for CD4(+) T cell mediated ocular surface disease in experimental dry eye disease.Interleukin-17 in various ocular surface inflammatory diseases Changes of Ocular Surface and the Inflammatory Response in a Rabbit Model of Short-Term Exposure Keratopathy.Association of a NOD2 gene polymorphism and T-helper 17 cells with presumed ocular toxoplasmosis.An immunoprotective privilege of corneal epithelial stem cells against Th17 inflammatory stress by producing glial cell-derived neurotrophic factor.Autoimmunity at the ocular surface: pathogenesis and regulation.Inflammatory Response to Lipopolysaccharide on the Ocular Surface in a Murine Dry Eye Model.Galectin-1-mediated suppression of Pseudomonas aeruginosa-induced corneal immunopathology.T helper cytokines in dry eye disease Pollen/TLR4 Innate Immunity Signaling Initiates IL-33/ST2/Th2 Pathways in Allergic Inflammation.Pivotal roles of T-helper 17-related cytokines, IL-17, IL-22, and IL-23, in inflammatory diseases.MUC1/A and MUC1/B splice variants differentially regulate inflammatory cytokine expression Peripheral Ulcerative Keratitis Associated with Autoimmune Disease: Pathogenesis and Treatment.The Pathophysiology of Dry Eye Disease: What We Know and Future Directions for Research.

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Induction of Th17 differentiation by corneal epithelial-derived cytokines

http://www.wikidata.org/entity/Q37462911

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on January 2010

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Induction of Th17 differentiation by corneal epithelial-derived cytokines

@en

Induction of Th17 differentiation by corneal epithelial-derived cytokines.

@nl

type

label

Induction of Th17 differentiation by corneal epithelial-derived cytokines

@en

Induction of Th17 differentiation by corneal epithelial-derived cytokines.

@nl

prefLabel

Induction of Th17 differentiation by corneal epithelial-derived cytokines

@en

Induction of Th17 differentiation by corneal epithelial-derived cytokines.

@nl

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Journal of Cellular Physiology

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Induction of Th17 differentiation by corneal epithelial-derived cytokines

@en

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Cintia S De Paiva

http://www.w3.org/2001/XMLSchema#string

De-Quan Li

http://www.w3.org/2001/XMLSchema#string

Fang Bian

http://www.w3.org/2001/XMLSchema#string

Michael Stern

http://www.w3.org/2001/XMLSchema#string

Ping Ma

http://www.w3.org/2001/XMLSchema#string

Stephen C Pflugfelder

http://www.w3.org/2001/XMLSchema#string

Xiaofen Zheng

http://www.w3.org/2001/XMLSchema#string

P2860

Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

The biological functions of T helper 17 cell effector cytokines in inflammation

The differentiation of human T(H)-17 cells requires transforming growth factor-beta and induction of the nuclear receptor RORgammat

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Transforming growth factor-beta induces development of the T(H)17 lineage

Interleukin-21: a modulator of lymphoid proliferation, apoptosis and differentiation

STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

TH1 and TH2 cells: different patterns of lymphokine secretion lead to different functional properties

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95-102

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scholarly article

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10.1002/JCP.21926

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1

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222

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2010-01-01T00:00:00Z

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26804670

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19746420

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2789678

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