Long-term exposure to concentrated ambient PM2.5 increases mouse blood pressure through abnormal activation of the sympathetic nervous system: a role for hypothalamic inflammation.
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Ambient particulate matter exposure and cardiovascular diseases: a focus on progenitor and stem cellsInfluencing Factors of Thermogenic Adipose Tissue ActivityPotential Harmful Effects of PM2.5 on Occurrence and Progression of Acute Coronary Syndrome: Epidemiology, Mechanisms, and Prevention MeasuresXenobiotic pulmonary exposure and systemic cardiovascular response via neurological linksCombined effects of exposure to dim light at night and fine particulate matter on C3H/HeNHsd miceThe preferential accumulation of heavy metals in different tissues following frequent respiratory exposure to PM2.5 in ratsEnvironmental stressors and cardio-metabolic disease: part II-mechanistic insights.Extreme Air Pollution Conditions Adversely Affect Blood Pressure and Insulin Resistance: The Air Pollution and Cardiometabolic Disease Study.Prenatal and postnatal mothering by diesel exhaust PM2.5-exposed dams differentially program mouse energy metabolismProgramming of mouse obesity by maternal exposure to concentrated ambient fine particlesPM2.5 and Diabetes and Hypertension Incidence in the Black Women's Health Study.Air pollution and adverse cardiac remodeling: clinical effects and basic mechanisms.Exposure to concentrated ambient particulate matter induces reversible increase of heart weight in spontaneously hypertensive rats.Exposure to air pollution near a steel plant is associated with reduced heart rate variability: a randomised crossover study.Basic mechanisms for adverse cardiovascular events associated with air pollution.Long-Term Exposure to NO2 and Ozone and Hypertension Incidence in the Black Women's Health Study.NF-κB-regulated microRNA-574-5p underlies synaptic and cognitive impairment in response to atmospheric PM2.5 aspiration.IRE1α/XBP1s branch of UPR links HIF1α activation to mediate ANGII-dependent endothelial dysfunction under particulate matter (PM) 2.5 exposure.Effect of Particulate Matter Air Pollution on Cardiovascular Oxidative Stress Pathways.Long-Term Exposure of Fine Particulate Matter Causes Hypertension by Impaired Renal D1 Receptor-Mediated Sodium Excretion via Upregulation of G-Protein-Coupled Receptor Kinase Type 4 Expression in Sprague-Dawley Rats.Exposure to Concentrated Ambient PM2.5 Compromises Spermatogenesis in a Mouse Model: Role of Suppression of Hypothalamus-Pituitary-Gonads Axis.Comparative cardiopulmonary effects of particulate matter- and ozone-enhanced smog atmospheres in mice.Inactivation of TNF/LT locus alters mouse metabolic response to concentrated ambient PM2.5.Short-Term Blood Pressure Responses to Ambient Fine Particulate Matter Exposures at the Extremes of Global Air Pollution Concentrations.Crucial role of pro-inflammatory cytokines from respiratory tract upon PM2.5 exposure in causing the BMSCs differentiation in cells and animals.Concentrated Ambient PM2.5-Induced Inflammation and Endothelial Dysfunction in a Murine Model of Neural IKK2 Deficiency.PM2.5 exposure aggravates oligomeric amyloid beta-induced neuronal injury and promotes NLRP3 inflammasome activation in an in vitro model of Alzheimer's disease.Ambient fine particulate matter exposure induces reversible cardiac dysfunction and fibrosis in juvenile and older female mice.County-level air quality and the prevalence of diagnosed chronic kidney disease in the US Medicare population
P2860
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P2860
Long-term exposure to concentrated ambient PM2.5 increases mouse blood pressure through abnormal activation of the sympathetic nervous system: a role for hypothalamic inflammation.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
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name
Long-term exposure to concentr ...... for hypothalamic inflammation.
@en
Long-term exposure to concentr ...... for hypothalamic inflammation.
@nl
type
label
Long-term exposure to concentr ...... for hypothalamic inflammation.
@en
Long-term exposure to concentr ...... for hypothalamic inflammation.
@nl
prefLabel
Long-term exposure to concentr ...... for hypothalamic inflammation.
@en
Long-term exposure to concentr ...... for hypothalamic inflammation.
@nl
P2093
P2860
P356
P1476
Long-term exposure to concentr ...... for hypothalamic inflammation
@en
P2093
Catherine Spino
Dongyao Liu
Jack R Harkema
Jinzhuo Zhao
Masako Morishita
Minjie Chen
Qinghua Sun
Robert D Brook
Sanjay Rajagopalan
Xiaohua Xu
P2860
P356
10.1289/EHP.1307151
P50
P577
2013-11-15T00:00:00Z