Genetic control of the alternative pathway of complement in humans and age-related macular degeneration - Wikidata - wikimedia - TriplyDB

Genetic control of the alternative pathway of complement in humans and age-related macular degeneration

Genetic control of the alternative pathway of complement in humans and age-related macular degeneration

http://www.wikidata.org/entity/Q37471769

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Inflammation and its role in age-related macular degeneration The complotype: dictating risk for inflammation and infection Inhibiting Alternative Pathway Complement Activation by Targeting the Factor D Exosite Fit for the Eye: Aptamers in Ocular Disorders Estimating the predictive ability of genetic risk models in simulated data based on published results from genome-wide association studies.Tissue-specific host recognition by complement factor H is mediated by differential activities of its glycosaminoglycan-binding regions.Therapeutic effect of oral bisphosphonates on choroidal neovascularization in the human eye.A common complement C3 variant is associated with protection against wet age-related macular degeneration in a Japanese population Systemic human CR2-targeted complement alternative pathway inhibitor ameliorates mouse laser-induced choroidal neovascularization.Complement system in pathogenesis of AMD: dual player in degeneration and protection of retinal tissue.Genome-wide association analyses of genetic, phenotypic, and environmental risks in the age-related eye disease study.Zinc supplementation inhibits complement activation in age-related macular degeneration.The significance of the complement system for the pathogenesis of age-related macular degeneration - current evidence and translation into clinical application The role of macrophage class a scavenger receptors in a laser-induced murine choroidal neovascularization model.The effect of lutein supplementation on blood plasma levels of complement factor D, C5a and C3d.Complement system dysregulation and inflammation in the retinal pigment epithelium of a mouse model for Stargardt macular degeneration.Common polymorphisms in C3, factor B, and factor H collaborate to determine systemic complement activity and disease risk Impact of the common genetic associations of age-related macular degeneration upon systemic complement component C3d levels Interleukin-17 retinotoxicity is prevented by gene transfer of a soluble interleukin-17 receptor acting as a cytokine blocker: implications for age-related macular degeneration.Complement component C5a promotes expression of IL-22 and IL-17 from human T cells and its implication in age-related macular degeneration.Copy number variation in the complement factor H-related genes and age-related macular degeneration CFH Y402H polymorphism is associated with elevated vitreal GM-CSF and choroidal macrophages in the postmortem human eye.Systemic and Ocular Long Pentraxin 3 in Patients with Age-Related Macular Degeneration.Age-Related Macular Degeneration: A Disease of Systemic or Local Complement Dysregulation?Small-molecule factor D inhibitors targeting the alternative complement pathway.Lutein Leads to a Decrease of Factor D Secretion by Cultured Mature Human Adipocytes.Increased plasma mannose binding lectin levels are associated with bronchiolitis obliterans after lung transplantation.Prevalence of age-related macular degeneration among the elderly.Progress and Trends in Complement Therapeutics Complement factor H Val62Ile variant and risk of age-related macular degeneration: a meta-analysis.Role of heparan sulfate in ocular diseases.Genetic variation in complement regulators and susceptibility to age-related macular degeneration.Gene expression changes in aging retinal microglia: relationship to microglial support functions and regulation of activation.Functional effect of Saffron supplementation and risk genotypes in early age-related macular degeneration: a preliminary report Recent developments in the treatment of age-related macular degeneration.The role of complement in age-related macular degeneration: heparan sulphate, a ZIP code for complement factor H?Application of quantile regression to recent genetic and -omic studies.The role of glial cells and the complement system in retinal diseases and Alzheimer's disease: common neural degeneration mechanisms.Drugs in Phase II clinical trials for the treatment of age-related macular degeneration.Inhibition of the alternative complement pathway preserves photoreceptors after retinal injury.

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Genetic control of the alternative pathway of complement in humans and age-related macular degeneration

http://www.wikidata.org/entity/Q37471769

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on January 2010

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Genetic control of the alterna ...... e-related macular degeneration

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Genetic control of the alterna ...... -related macular degeneration.

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Genetic control of the alterna ...... e-related macular degeneration

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Genetic control of the alterna ...... -related macular degeneration.

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Genetic control of the alterna ...... e-related macular degeneration

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Genetic control of the alterna ...... -related macular degeneration.

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Human Molecular Genetics

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Genetic control of the alterna ...... e-related macular degeneration

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Albert O Edwards

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Beatrix Pollok-Kopp

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Euijung Ryu

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Hendrik P Scholl

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Katharina E Schmid-Kubista

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Keith H Baratz

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Kent R Bailey

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Laura A Hecker

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Martin Oppermann

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Peter Charbel Issa

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P2860

Susceptibility genes for age-related maculopathy on chromosome 10q26.

Complement factor H polymorphism in age-related macular degeneration

Risk factors for the incidence of Advanced Age-Related Macular Degeneration in the Age-Related Eye Disease Study (AREDS) AREDS report no. 19

Structural basis for complement factor H linked age-related macular degeneration

Complement. First of two parts

Complement factor H variant increases the risk of age-related macular degeneration

Systemic complement activation in age-related macular degeneration

Formation of the initial C3 convertase of the alternative complement pathway. Acquisition of C3b-like activities by spontaneous hydrolysis of the putative thioester in native C3

Complement factor H polymorphism and age-related macular degeneration

Estimation of systemic complement C3 activity in age-related macular degeneration.

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209-215

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scholarly article

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10.1093/HMG/DDP472

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1

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19

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Nirubol Tosakulwong

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2010-01-01T00:00:00Z

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26891459

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19825847

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2792151

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