An N-Terminal Missense Mutation in STX11 Causative of FHL4 Abrogates Syntaxin-11 Binding to Munc18-2.
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Analysis of familial hemophagocytic lymphohistiocytosis type 4 (FHL-4) mutant proteins reveals that S-acylation is required for the function of syntaxin 11 in natural killer cellsMolecular mechanisms regulating cytotoxic lymphocyte development and function, and their associations to human diseasesFamilial hemophagocytic lymphohistiocytosis: when rare diseases shed light on immune system functioning.A unique SNARE machinery for exocytosis of cytotoxic granules and platelets granules.SM protein Munc18-2 facilitates transition of Syntaxin 11-mediated lipid mixing to complete fusion for T-lymphocyte cytotoxicity.
P2860
An N-Terminal Missense Mutation in STX11 Causative of FHL4 Abrogates Syntaxin-11 Binding to Munc18-2.
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name
An N-Terminal Missense Mutatio ...... ntaxin-11 Binding to Munc18-2.
@en
An N-Terminal Missense Mutatio ...... ntaxin-11 Binding to Munc18-2.
@nl
type
label
An N-Terminal Missense Mutatio ...... ntaxin-11 Binding to Munc18-2.
@en
An N-Terminal Missense Mutatio ...... ntaxin-11 Binding to Munc18-2.
@nl
prefLabel
An N-Terminal Missense Mutatio ...... ntaxin-11 Binding to Munc18-2.
@en
An N-Terminal Missense Mutatio ...... ntaxin-11 Binding to Munc18-2.
@nl
P2093
P2860
P50
P356
P1476
An N-Terminal Missense Mutatio ...... yntaxin-11 Binding to Munc18-2
@en
P2093
Ahmed Naqvi
Marie Meeths
Martha-Lena Müller
Miriam Entesarian
Stephanie M Wood
P2860
P356
10.3389/FIMMU.2013.00515
P577
2014-01-14T00:00:00Z