Hypothalamic PI3K and MAPK differentially mediate regional sympathetic activation to insulin. - Wikidata - wikimedia - TriplyDB

Hypothalamic PI3K and MAPK differentially mediate regional sympathetic activation to insulin.

Hypothalamic PI3K and MAPK differentially mediate regional sympathetic activation to insulin.

http://www.wikidata.org/entity/Q37486718

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Leptin and insulin stimulation of signalling pathways in arcuate nucleus neurones: PI3K dependent actin reorganization and KATP channel activation Brain Insulin Resistance at the Crossroads of Metabolic and Cognitive Disorders in Humans Emerging role of the brain in the homeostatic regulation of energy and glucose metabolism The brain and brown fat Obesity and adipokines: effects on sympathetic overactivity Brain Regulation of Energy Metabolism SIRT1 deacetylase in POMC neurons is required for homeostatic defenses against diet-induced obesity Central resistin enhances renal sympathetic nerve activity via phosphatidylinositol 3-kinase but reduces the activity to brown adipose tissue via extracellular signal-regulated kinase 1/2 Neuroadrenergic dysfunction along the diabetes continuum: a comparative study in obese metabolic syndrome subjects.Central neural regulation of brown adipose tissue thermogenesis and energy expenditure Divergent regulation of energy expenditure and hepatic glucose production by insulin receptor in agouti-related protein and POMC neurons Is insulin the new intermittent hypoxia?Obesity-Induced Hypertension: Brain Signaling Pathways.Influence of endurance training on central sympathetic outflow to skeletal muscle in response to a mixed meal.Glutamatergic receptor activation in the rostral ventrolateral medulla mediates the sympathoexcitatory response to hyperinsulinemia Differential effects of insulin on sympathetic nerve activity in agouti obese mice.Hypothalamic AMPK and fatty acid metabolism mediate thyroid regulation of energy balance.Obesity and leptin resistance: distinguishing cause from effect.Excess of nerve growth factor in the ovary causes a polycystic ovary-like syndrome in mice, which closely resembles both reproductive and metabolic aspects of the human syndrome.Glucocorticoids attenuate the central sympathoexcitatory actions of insulin.Brain insulin controls adipose tissue lipolysis and lipogenesis.Glutamate receptors in the hypothalamic paraventricular nucleus contribute to insulin-induced sympathoexcitation The functional role of PI3K in maintenance of blood pressure and baroreflex suppression in (mRen2)27 and mRen2.Lewis rat.The role of hypothalamic mTORC1 signaling in insulin regulation of food intake, body weight, and sympathetic nerve activity in male mice.Stimulatory effect of insulin on glucose uptake by muscle involves the central nervous system in insulin-sensitive mice Central nervous system regulation of brown adipose tissue.Leptin and insulin act on POMC neurons to promote the browning of white fat.Central effects of humanin on hepatic triglyceride secretion.The central melanocortin system directly controls peripheral lipid metabolism.Mitogen-activated protein kinase contributes to lipopolysaccharide-induced activation of corticotropin-releasing hormone synthesizing neurons in the hypothalamic paraventricular nucleus.Central insulin action regulates peripheral glucose and fat metabolism in mice.Regulation of Blood Pressure, Appetite, and Glucose by Leptin After Inactivation of Insulin Receptor Substrate 2 Signaling in the Entire Brain or in Proopiomelanocortin Neurons.Sympathetic response to insulin is mediated by melanocortin 3/4 receptors in the hypothalamic paraventricular nucleus Sympathoexcitation associated with Renin-Angiotensin system in metabolic syndrome.Cardiovascular Regulation by the Arcuate Nucleus of the Hypothalamus: Neurocircuitry and Signaling Systems.Arcuate nucleus injection of an anti-insulin affibody prevents the sympathetic response to insulin Amylin acts in the central nervous system to increase sympathetic nerve activity Pressor recovery after acute stress is impaired in high fructose-fed Lean Zucker rats.Hypothalamic roles of mTOR complex I: integration of nutrient and hormone signals to regulate energy homeostasis Hypothalamic ERK mediates the anorectic and thermogenic sympathetic effects of leptin

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Hypothalamic PI3K and MAPK differentially mediate regional sympathetic activation to insulin.

http://www.wikidata.org/entity/Q37486718

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on September 2004

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Hypothalamic PI3K and MAPK dif ...... athetic activation to insulin.

@en

Hypothalamic PI3K and MAPK dif ...... athetic activation to insulin.

@nl

type

label

Hypothalamic PI3K and MAPK dif ...... athetic activation to insulin.

@en

Hypothalamic PI3K and MAPK dif ...... athetic activation to insulin.

@nl

prefLabel

Hypothalamic PI3K and MAPK dif ...... athetic activation to insulin.

@en

Hypothalamic PI3K and MAPK dif ...... athetic activation to insulin.

@nl

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Journal of Clinical Investigation

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Hypothalamic PI3K and MAPK dif ...... athetic activation to insulin.

@en

P2093

Allyn L Mark

http://www.w3.org/2001/XMLSchema#string

Donald A Morgan

http://www.w3.org/2001/XMLSchema#string

Gina M Morgan

http://www.w3.org/2001/XMLSchema#string

William G Haynes

http://www.w3.org/2001/XMLSchema#string

Xuebo Liu

http://www.w3.org/2001/XMLSchema#string

P2860

Insulin signalling and the regulation of glucose and lipid metabolism

Specificity and mechanism of action of some commonly used protein kinase inhibitors

PD 098059 is a specific inhibitor of the activation of mitogen-activated protein kinase kinase in vitro and in vivo

Identification of a novel inhibitor of mitogen-activated protein kinase kinase

Spatial compartmentalization of signal transduction in insulin action.

Insulin-mediated sympathetic stimulation: role in the pathogenesis of obesity-related hypertension (or, how insulin affects blood pressure, and why).

Role of insulin and insulin receptor in learning and memory.

Hyperinsulinemia produces both sympathetic neural activation and vasodilation in normal humans

Abnormal sympathetic overactivity evoked by insulin in the skeletal muscle of patients with essential hypertension

Insulin and leptin as adiposity signals.

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652-658

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scholarly article

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10.1172/JCI21737

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5

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114

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Curt D. Sigmund

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2004-09-01T00:00:00Z

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