Potentiation of GluN2C/D NMDA receptor subtypes in the amygdala facilitates the retention of fear and extinction learning in mice.
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Pharmacology of cognitive enhancers for exposure-based therapy of fear, anxiety and trauma-related disordersMechanisms to medicines: elucidating neural and molecular substrates of fear extinction to identify novel treatments for anxiety disordersd-Cycloserine augmentation of cognitive remediation in schizophrenia.GluN2C/GluN2D subunit-selective NMDA receptor potentiator CIQ reverses MK-801-induced impairment in prepulse inhibition and working memory in Y-maze test in mice.Glutamate, GABA, and glutamine are synchronously upregulated in the mouse lateral septum during the postpartum period.Hippocampal Wnt3a is Necessary and Sufficient for Contextual Fear Memory Acquisition and Consolidation.Specific Roles of NMDA Receptor Subunits in Mental Disorders.Glutamatergic regulation of cognition and functional brain connectivity: insights from pharmacological, genetic and translational schizophrenia research.D-cycloserine in Schizophrenia: New Strategies for Improving Clinical Outcomes by Enhancing Plasticity.Neural circuits involved in the renewal of extinguished fear.Cocaine Experience Enhances Thalamo-Accumbens N-Methyl-D-Aspartate Receptor Function.Allosteric modulation of GluN2C/GluN2D-containing NMDA receptors bidirectionally modulates dopamine release: implication for Parkinson's disease.The bioactive protein-ligand conformation of GluN2C-selective positive allosteric modulators bound to the NMDA receptor.CIQ, a positive allosteric modulator of GluN2C/D-containing N-methyl-d-aspartate receptors, rescues striatal synaptic plasticity deficit in a mouse model of Parkinson's disease.A Novel Negative Allosteric Modulator Selective for GluN2C/2D-Containing NMDA Receptors Inhibits Synaptic Transmission in Hippocampal Interneurons.NMDA Receptors in the Central Nervous System.The Structure-Activity Relationship of a Tetrahydroisoquinoline Class of N-Methyl-d-Aspartate Receptor Modulators that Potentiates GluN2B-Containing N-Methyl-d-Aspartate Receptors.Identification of AICP as a GluN2C-Selective N-Methyl-d-Aspartate Receptor Superagonist at the GluN1 Glycine Site.An NMDAR positive and negative allosteric modulator series share a binding site and are interconverted by methyl groups.How does binding of agonist ligands control intrinsic molecular dynamics in human NMDA receptors?
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P2860
Potentiation of GluN2C/D NMDA receptor subtypes in the amygdala facilitates the retention of fear and extinction learning in mice.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 06 September 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Potentiation of GluN2C/D NMDA ...... d extinction learning in mice.
@en
Potentiation of GluN2C/D NMDA ...... d extinction learning in mice.
@nl
type
label
Potentiation of GluN2C/D NMDA ...... d extinction learning in mice.
@en
Potentiation of GluN2C/D NMDA ...... d extinction learning in mice.
@nl
prefLabel
Potentiation of GluN2C/D NMDA ...... d extinction learning in mice.
@en
Potentiation of GluN2C/D NMDA ...... d extinction learning in mice.
@nl
P2093
P2860
P356
P1476
Potentiation of GluN2C/D NMDA ...... nd extinction learning in mice
@en
P2093
Alpa Khatri
Scott A Heldt
Stephen F Traynelis
P2860
P2888
P304
P356
10.1038/NPP.2013.241
P407
P50
P577
2013-09-06T00:00:00Z