Loss of β-adrenergic-stimulated phosphorylation of CaV1.2 channels on Ser1700 leads to heart failure
about
Proteolytic cleavage and PKA phosphorylation of α1C subunit are not required for adrenergic regulation of CaV1.2 in the heart.Identification of a novel cAMP dependent protein kinase A phosphorylation site on the human cardiac calcium channel.Protein kinase A regulates C-terminally truncated CaV 1.2 in Xenopus oocytes: roles of N- and C-termini of the α1C subunit.The AKAP Cypher/Zasp contributes to β-adrenergic/PKA stimulation of cardiac CaV1.2 calcium channels.Voltage-gated calcium channels: their discovery, function and importance as drug targets
P2860
Loss of β-adrenergic-stimulated phosphorylation of CaV1.2 channels on Ser1700 leads to heart failure
description
2016 nî lūn-bûn
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name
Loss of β-adrenergic-stimulate ...... Ser1700 leads to heart failure
@en
Loss of β-adrenergic-stimulate ...... er1700 leads to heart failure.
@nl
type
label
Loss of β-adrenergic-stimulate ...... Ser1700 leads to heart failure
@en
Loss of β-adrenergic-stimulate ...... er1700 leads to heart failure.
@nl
prefLabel
Loss of β-adrenergic-stimulate ...... Ser1700 leads to heart failure
@en
Loss of β-adrenergic-stimulate ...... er1700 leads to heart failure.
@nl
P2093
P2860
P356
P1476
Loss of β-adrenergic-stimulate ...... Ser1700 leads to heart failure
@en
P2093
Horacio O de la Iglesia
Linghai Yang
Nastassya West
Ruth E Westenbroek
William A Catterall
P2860
P304
E7976-E7985
P356
10.1073/PNAS.1617116113
P407
P577
2016-11-18T00:00:00Z