Mice lacking natural killer T cells are more susceptible to metabolic alterations following high fat diet feeding.
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Molecular Pathogenesis of NASHAdipose-tissue and intestinal inflammation - visceral obesity and creeping fatLymphocytes in atherosclerosis.Combined B, T and NK Cell Deficiency Accelerates Atherosclerosis in BALB/c MiceAnimal Models Correlating Immune Cells for the Development of NAFLD/NASH.Choline Deficiency Causes Colonic Type II Natural Killer T (NKT) Cell Loss and Alleviates Murine Colitis under Type I NKT Cell Deficiency.Changes in peripheral blood immune cell composition in osteoarthritis.Mice subjected to aP2-Cre mediated ablation of microsomal triglyceride transfer protein are resistant to high fat diet induced obesityHomeostasis of IL-15 dependent lymphocyte subsets in the liver.Innate Immunity and Inflammation in NAFLD/NASH.Natural killer T cells in atherosclerosis.Oral administration of a non-absorbable plant cell-expressed recombinant anti-TNF fusion protein induces immunomodulatory effects and alleviates nonalcoholic steatohepatitis.Unusual Suspects in the Development of Obesity-Induced Inflammation and Insulin Resistance: NK cells, iNKT cells, and ILCs.Endogenous lipid antigens for invariant Natural Killer T cells hold the reins in adipose tissue homeostasis.Iron overload results in hepatic oxidative stress, immune cell activation, and hepatocellular ballooning injury, leading to nonalcoholic steatohepatitis in genetically obese mice.Deficiency of Invariant Natural Killer T Cells Does Not Protect Against Obesity but Exacerbates Atherosclerosis in Ldlr-/- Mice.TCRα-TCRβ pairing controls recognition of CD1d and directs the development of adipose NKT cells.The Immune System in Obesity: Developing Paradigms Amidst Inconvenient Truths.M2-specific reduction of CD1d switches NKT cell-mediated immune responses and triggers metaflammation in adipose tissue.Physical Inactivity and Unhealthy Metabolic Status Are Associated with Decreased Natural Killer Cell Activity.Molecular Pathogenesis of Nonalcoholic Steatohepatitis- (NASH-) Related Hepatocellular Carcinoma
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Mice lacking natural killer T cells are more susceptible to metabolic alterations following high fat diet feeding.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 20 January 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Mice lacking natural killer T ...... llowing high fat diet feeding.
@en
Mice lacking natural killer T ...... llowing high fat diet feeding.
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type
label
Mice lacking natural killer T ...... llowing high fat diet feeding.
@en
Mice lacking natural killer T ...... llowing high fat diet feeding.
@nl
prefLabel
Mice lacking natural killer T ...... llowing high fat diet feeding.
@en
Mice lacking natural killer T ...... llowing high fat diet feeding.
@nl
P2093
P2860
P1433
P1476
Mice lacking natural killer T ...... llowing high fat diet feeding.
@en
P2093
Becky A De La Houssaye
Brittany V Martin-Murphy
Cynthia Ju
Jacob E Friedman
Timothy P Reilly
P2860
P304
P356
10.1371/JOURNAL.PONE.0080949
P407
P577
2014-01-20T00:00:00Z