MiR-155-mediated loss of C/EBPβ shifts the TGF-β response from growth inhibition to epithelial-mesenchymal transition, invasion and metastasis in breast cancer.
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Mechanisms of TGFβ-Induced Epithelial-Mesenchymal TransitionEpithelial-Mesenchymal Transition and Breast CancerTransforming growth factor β as regulator of cancer stemness and metastasisSwitching Roles of TGF-β in Cancer Development: Implications for Therapeutic Target and Biomarker StudiesSignal transducer and activator of transcription 3 in myeloid-derived suppressor cells: an opportunity for cancer therapyA genome-scale screen reveals context-dependent ovarian cancer sensitivity to miRNA overexpressionMicroRNA-34a targets epithelial to mesenchymal transition-inducing transcription factors (EMT-TFs) and inhibits breast cancer cell migration and invasionMicroRNA-155 expression is independently predictive of outcome in chordoma.MiR-155 up-regulated by TGF-β promotes epithelial-mesenchymal transition, invasion and metastasis of human hepatocellular carcinoma cells in vitro.miR-155 induced transcriptome changes in the MCF-7 breast cancer cell line leads to enhanced mitogen activated protein kinase signalingMCRS1 overexpression, which is specifically inhibited by miR-129*, promotes the epithelial-mesenchymal transition and metastasis in non-small cell lung cancer.C/EBPβ expression is an independent predictor of overall survival in breast cancer patients by MHCII/CD4-dependent mechanism of metastasis formation.Feasibility of urinary microRNA detection in breast cancer patients and its potential as an innovative non-invasive biomarkermiR-191 promotes tumorigenesis of human colorectal cancer through targeting C/EBPβ.MicroRNA-155 Deficiency Attenuates Liver Steatosis and Fibrosis without Reducing Inflammation in a Mouse Model of Steatohepatitis.Inhibition of MiR-155 suppresses cell migration in nasopharyngeal carcinoma through targeting ZDHHC2.Identification of microRNAs specific for epithelial cell adhesion molecule-positive tumor cells in hepatocellular carcinoma.OVOL guides the epithelial-hybrid-mesenchymal transition.Reprogramming during epithelial to mesenchymal transition under the control of TGFβOrosomucoid 2 inhibits tumor metastasis and is upregulated by CCAAT/enhancer binding protein β in hepatocellular carcinomas.The candidate oncogene (MCRS1) promotes the growth of human lung cancer cells via the miR-155-Rb1 pathway.Morusin suppresses breast cancer cell growth in vitro and in vivo through C/EBPβ and PPARγ mediated lipoapoptosis.MiRNA-155 mediates TAM resistance by modulating SOCS6-STAT3 signalling pathway in breast cancer.Regulators of genetic risk of breast cancer identified by integrative network analysis.Double-stranded microRNA mimics can induce length- and passenger strand-dependent effects in a cell type-specific manner.Overexpression of miR-155 promotes the proliferation and invasion of oral squamous carcinoma cells by regulating BCL6/cyclin D2NF-κB-driven suppression of FOXO3a contributes to EGFR mutation-independent gefitinib resistance.Role of TGF-β in breast cancer bone metastases.Id4 promotes cell proliferation in hepatocellular carcinoma.miRNAs and their role in the correlation between schizophrenia and cancer (Review).Targeting the microRNA-regulating DNA damage/repair pathways in cancer.Role of miR-155 in drug resistance of breast cancer.miRNA therapy targeting cancer stem cells: a new paradigm for cancer treatment and prevention of tumor recurrence.Recent advances of microRNA-based molecular diagnostics to reduce false-positive lung cancer imaging.Does miRNA-155 Promote Cyclooxygenase-2 Expression in Cancer?Crosstalk between TGF-β signaling and miRNAs in breast cancer metastasis.Molecular insights into tumour metastasis: tracing the dominant events.The role of microRNAs in bone metastasis.Mucin1 promotes the migration and invasion of hepatocellular carcinoma cells via JNK-mediated phosphorylation of Smad2 at the C-terminal and linker regions.C/EBPβ Mediates TNF-α-Induced Cancer Cell Migration by Inducing MMP Expression Dependent on p38 MAPK.
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MiR-155-mediated loss of C/EBPβ shifts the TGF-β response from growth inhibition to epithelial-mesenchymal transition, invasion and metastasis in breast cancer.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 19 August 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
MiR-155-mediated loss of C/EBP ...... d metastasis in breast cancer.
@en
MiR-155-mediated loss of C/EBP ...... d metastasis in breast cancer.
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type
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MiR-155-mediated loss of C/EBP ...... d metastasis in breast cancer.
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MiR-155-mediated loss of C/EBP ...... d metastasis in breast cancer.
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prefLabel
MiR-155-mediated loss of C/EBP ...... d metastasis in breast cancer.
@en
MiR-155-mediated loss of C/EBP ...... d metastasis in breast cancer.
@nl
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MiR-155-mediated loss of C/EBP ...... d metastasis in breast cancer.
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P2093
J Johansson
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10.1038/ONC.2013.322
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P577
2013-08-19T00:00:00Z