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Chemokines and Heart Disease: A Network Connecting Cardiovascular Biology to Immune and Autonomic Nervous SystemsCardiac myocyte diversity and a fibroblast network in the junctional region of the zebrafish heart revealed by transmission and serial block-face scanning electron microscopyTGF-β1, released by myofibroblasts, differentially regulates transcription and function of sodium and potassium channels in adult rat ventricular myocytesBNIP3L promotes cardiac fibrosis in cardiac fibroblasts through [Ca2+]i-TGF-β-Smad2/3 pathway.Decreased fibrocyte number is associated with atherosclerotic plaque instability in man.Inducible nitric oxide synthase in heart tissue and nitric oxide in serum of Trypanosoma cruzi-infected rhesus monkeys: association with heart injury.Inhibitory effects of C-type natriuretic peptide on the differentiation of cardiac fibroblasts, and secretion of monocyte chemoattractant protein-1 and plasminogen activator inhibitor-1.The cardiokine story unfolds: ischemic stress-induced protein secretion in the heartProtease activated receptor-2 contributes to heart failure.Aging and Cardiac Fibrosis.Coronary artery ligation and intramyocardial injection in a murine model of infarction.Biomaterial-mediated modification of the local inflammatory environment.Tissue-nonspecific alkaline phosphatase as a target of sFRP2 in cardiac fibroblasts.Diabetes-associated cardiac fibrosis: Cellular effectors, molecular mechanisms and therapeutic opportunities.Plakophilin-2 loss promotes TGF-β1/p38 MAPK-dependent fibrotic gene expression in cardiomyocytes.Unique mechanistic insights into the beneficial effects of soluble epoxide hydrolase inhibitors in the prevention of cardiac fibrosis.Cardiac-Secreted Factors as Peripheral Metabolic Regulators and Potential Disease Biomarkers.The pathogenesis of cardiac fibrosis.Associations between fibrocytes and postcontrast myocardial T1 times in hypertrophic cardiomyopathy.Intermedin1-53 Protects Against Myocardial Fibrosis by Inhibiting Endoplasmic Reticulum Stress and Inflammation Induced by Homocysteine in Apolipoprotein E-Deficient Mice.Secreted Frizzled-related protein 2 as a target in antifibrotic therapeutic intervention.Lack of ABCG2 Leads to Biventricular Dysfunction and Remodeling in Response to HypoxiaExtracellular matrix roles during cardiac repair.Stem cell transplantation as a therapy for cardiac fibrosis.Involvement of monocytes/macrophages as key factors in the development and progression of cardiovascular diseases.Non-coding RNAs as direct and indirect modulators of epigenetic mechanism regulation of cardiac fibrosis.Relevance of mouse models of cardiac fibrosis and hypertrophy in cardiac research.Aristoyunnolin H attenuates extracellular matrix secretion in cardiac fibroblasts by inhibiting calcium influx.CYP epoxygenase 2J2 prevents cardiac fibrosis by suppression of transmission of pro-inflammation from cardiomyocytes to macrophages.Sphingosine 1-Phosphate Receptor Modulator Fingolimod (FTY720) Attenuates Myocardial Fibrosis in Post-heterotopic Heart Transplantation.sFRP2 activates Wnt/β-catenin signaling in cardiac fibroblasts: differential roles in cell growth, energy metabolism, and extracellular matrix remodeling.Serum profiles of monocyte chemoattractant protein-1 as a biomarker for patients recovering from myocardial infarction.MEK1-induced physiological hypertrophy inhibits chronic post-myocardial infarction remodeling in mice.Fibroblasts and the extracellular matrix in right ventricular disease.Immune mechanisms in heart failure.A comparison of left and right atrial fibroblasts reveals different collagen production activity and stress-induced mitogen-activated protein kinase signalling in rats.Curcumin reduces cardiac fibrosis by inhibiting myofibroblast differentiation and decreasing transforming growth factor β1 and matrix metalloproteinase 9 / tissue inhibitor of metalloproteinase 1.Selective PDE5A inhibition with sildenafil rescues left ventricular dysfunction, inflammatory immune response and cardiac remodeling in angiotensin II-induced heart failure in vivo.In Silico Analysis of Differential Gene Expression in Three Common Rat Models of Diastolic Dysfunction.
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Chemokines and cardiac fibrosis.
@en
Chemokines and cardiac fibrosis.
@nl
type
label
Chemokines and cardiac fibrosis.
@en
Chemokines and cardiac fibrosis.
@nl
prefLabel
Chemokines and cardiac fibrosis.
@en
Chemokines and cardiac fibrosis.
@nl
P2860
P356
P1476
Chemokines and cardiac fibrosis
@en
P2093
Marcin Dobaczewski
P2860
P304
P356
10.2741/S33
P577
2009-06-01T00:00:00Z