The Ku protein complex interacts with YY1, is up-regulated in human heart failure, and represses alpha myosin heavy-chain gene expression.
about
Astrocyte elevated gene-1: recent insights into a novel gene involved in tumor progression, metastasis and neurodegenerationAstrocyte elevated gene-1 (AEG-1) is a target gene of oncogenic Ha-ras requiring phosphatidylinositol 3-kinase and c-MycKu proteins interact with activator protein-2 transcription factors and contribute to ERBB2 overexpression in breast cancer cell lines.The DNA repair complex Ku70/86 modulates Apaf1 expression upon DNA damageChromatin association of XRCC5/6 in the absence of DNA damage depends on the XPE gene product DDB2.Transgenic over-expression of YY1 induces pathologic cardiac hypertrophy in a sex-specific mannerDNA ends alter the molecular composition and localization of Ku multicomponent complexes.Discordant on/off switching of gene expression in myocytes during cardiac hypertrophy in vivo.Factors controlling cardiac myosin-isoform shift during hypertrophy and heart failureFunction of YY1 in Long-Distance DNA Interactions.Transcription factors in heart: promising therapeutic targets in cardiac hypertrophy.Host Proteins Ku and HMGA1 As Participants of HIV-1 Transcription.YY1 controls immunoglobulin class switch recombination and nuclear activation-induced deaminase levels.Physical interaction between TBX5 and MEF2C is required for early heart development.Reversible epigenetic modifications of the two cardiac myosin heavy chain genes during changes in expression.NF-kappaB regulation of YY1 inhibits skeletal myogenesis through transcriptional silencing of myofibrillar genes.Oxidative DNA damage in neurons: implication of ku in neuronal homeostasis and survival.Endothelin-stimulated human B-type natriuretic peptide gene expression is mediated by Yin Yang 1 in association with histone deacetylase 2.YY1 Expression Is Sufficient for the Maintenance of Cardiac Progenitor Cell State.Activation of SIRT1, a class III histone deacetylase, contributes to fructose feeding-mediated induction of the alpha-myosin heavy chain expression.Critical role of YY1 in cardiac morphogenesis.Shuttling of HDAC5 in H9C2 cells regulates YY1 function through CaMKIV/PKD and PP2A.
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The Ku protein complex interacts with YY1, is up-regulated in human heart failure, and represses alpha myosin heavy-chain gene expression.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on October 2004
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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The Ku protein complex interac ...... n heavy-chain gene expression.
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The Ku protein complex interac ...... n heavy-chain gene expression.
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The Ku protein complex interac ...... n heavy-chain gene expression.
@en
The Ku protein complex interac ...... n heavy-chain gene expression.
@nl
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The Ku protein complex interac ...... n heavy-chain gene expression.
@en
The Ku protein complex interac ...... n heavy-chain gene expression.
@nl
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The Ku protein complex interac ...... in heavy-chain gene expression
@en
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Carmen C Sucharov
M Benjamin Perryman
Michael Bristow
Stephen J Langer
Steve M Helmke
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P304
P356
10.1128/MCB.24.19.8705-8715.2004
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P577
2004-10-01T00:00:00Z