Bloom syndrome complex promotes FANCM recruitment to stalled replication forks and facilitates both repair and traverse of DNA interstrand crosslinks
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FANCD2 and DNA Damage.DNA damage response and cancer therapeutics through the lens of the Fanconi Anemia DNA repair pathway.RECQ-like helicases Sgs1 and BLM regulate R-loop-associated genome instability.DNA Damage as a Driver for Growth Delay: Chromosome Instability Syndromes with Intrauterine Growth Retardation.Fanconi Anemia Signaling and Cancer.
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Bloom syndrome complex promotes FANCM recruitment to stalled replication forks and facilitates both repair and traverse of DNA interstrand crosslinks
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 20 December 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Bloom syndrome complex promote ...... of DNA interstrand crosslinks
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Bloom syndrome complex promote ...... of DNA interstrand crosslinks.
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Bloom syndrome complex promote ...... of DNA interstrand crosslinks
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Bloom syndrome complex promote ...... of DNA interstrand crosslinks.
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Bloom syndrome complex promote ...... of DNA interstrand crosslinks
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Bloom syndrome complex promote ...... of DNA interstrand crosslinks.
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Bloom syndrome complex promote ...... of DNA interstrand crosslinks
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Jing Huang
Masamichi Ishiai
Michael Seidman
Mioko Ohzeki
Weidong Wang
Yongjiang Li
Zhijiang Yan
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10.1038/CELLDISC.2016.47
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2016-12-20T00:00:00Z