Differential requirements for VirB1 and VirB2 during Brucella abortus infection.
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Type IV secretion system of Brucella spp. and its effectorsVirB1* promotes T-pilus formation in the vir-Type IV secretion system of Agrobacterium tumefaciens.RNAi screen of endoplasmic reticulum-associated host factors reveals a role for IRE1alpha in supporting Brucella replication.Whole genome transcription profiling of Anaplasma phagocytophilum in human and tick host cells by tiling array analysisAn anomalous type IV secretion system in Rickettsia is evolutionarily conserved.Brucella melitensis VjbR and C12-HSL regulons: contributions of the N-dodecanoyl homoserine lactone signaling molecule and LuxR homologue VjbR to gene expression.An empirical strategy for characterizing bacterial proteomes across species in the absence of genomic sequences.Phylogenomics reveals a diverse Rickettsiales type IV secretion system.Brucella abortus virB12 is expressed during infection but is not an essential component of the type IV secretion systemEarly transcriptional responses of bovine chorioallantoic membrane explants to wild type, ΔvirB2 or ΔbtpB Brucella abortus infection.The Brucella suis type IV secretion system assembles in the cell envelope of the heterologous host Agrobacterium tumefaciens and increases IncQ plasmid pLS1 recipient competenceBiogenesis, architecture, and function of bacterial type IV secretion systemsThe predicted ABC transporter AbcEDCBA is required for type IV secretion system expression and lysosomal evasion by Brucella ovis.Deletion of znuA virulence factor attenuates Brucella abortus and confers protection against wild-type challengeEvaluation of protection afforded by Brucella abortus and Brucella melitensis unmarked deletion mutants exhibiting different rates of clearance in BALB/c mice.An in vivo high-throughput screening approach targeting the type IV secretion system component VirB8 identified inhibitors of Brucella abortus 2308 proliferation.Establishment of systemic Brucella melitensis infection through the digestive tract requires urease, the type IV secretion system, and lipopolysaccharide O antigenImpact of plasmids, including those encodingVirB4/D4 type IV secretion systems, on Salmonella enterica serovar Heidelberg virulence in macrophages and epithelial cellsNatural transformation of Myxococcus xanthus.The abcEDCBA-Encoded ABC Transporter and the virB Operon-Encoded Type IV Secretion System of Brucella ovis Are Critical for Intracellular Trafficking and Survival in Ovine Monocyte-Derived Macrophages.Quorum-sensing and BvrR/BvrS regulation, the type IV secretion system, cyclic glucans, and BacA in the virulence of Brucella ovis: similarities to and differences from smooth brucellaeThe small protein CydX is required for function of cytochrome bd oxidase in Brucella abortusVirB12 is a serological marker of Brucella infection in experimental and natural hosts.A lysozyme-like protein in Brucella abortus is involved in the early stages of intracellular replication.Inactivation of the type IV secretion system reduces the Th1 polarization of the immune response to Brucella abortus infection.VirB3 to VirB6 and VirB8 to VirB11, but not VirB7, are essential for mediating persistence of Brucella in the reticuloendothelial system.Sensing of bacterial type IV secretion via the unfolded protein response.Progress in Brucella vaccine development.Erythritol triggers expression of virulence traits in Brucella melitensis.Natural antibody contributes to host defense against an attenuated Brucella abortus virB mutant.The role of NLRP3 and AIM2 in inflammasome activation during Brucella abortus infection.Brucellosis and type IV secretion.RNA-seq reveals the critical role of CspA in regulating Brucella melitensis metabolism and virulence.Chronic Bacterial Pathogens: Mechanisms of Persistence.Identification of VceA and VceC, two members of the VjbR regulon that are translocated into macrophages by the Brucella type IV secretion system.Development and trial of vaccines against Brucella.Chronic Brucella Infection Induces Selective and Persistent Interferon Gamma-Dependent Alterations of Marginal Zone Macrophages in the Spleen.Restoring virulence to mutants lacking subunits of multiprotein machines: functional complementation of a Brucella virB5 mutant.Mice lacking components of adaptive immunity show increased Brucella abortus virB mutant colonization.Brucella requires a functional Type IV secretion system to elicit innate immune responses in mice.
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Differential requirements for VirB1 and VirB2 during Brucella abortus infection.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on September 2004
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Differential requirements for VirB1 and VirB2 during Brucella abortus infection.
@en
Differential requirements for VirB1 and VirB2 during Brucella abortus infection.
@nl
type
label
Differential requirements for VirB1 and VirB2 during Brucella abortus infection.
@en
Differential requirements for VirB1 and VirB2 during Brucella abortus infection.
@nl
prefLabel
Differential requirements for VirB1 and VirB2 during Brucella abortus infection.
@en
Differential requirements for VirB1 and VirB2 during Brucella abortus infection.
@nl
P2093
P2860
P1476
Differential requirements for VirB1 and VirB2 during Brucella abortus infection
@en
P2093
Andreas B den Hartigh
David Sondervan
Marjolein O Reinders
Niki Heuvelmans
Thomas A Ficht
Yao-Hui Sun
P2860
P304
P356
10.1128/IAI.72.9.5143-5149.2004
P407
P577
2004-09-01T00:00:00Z