Sodium transport is modulated by p38 kinase-dependent cross-talk between ENaC and Na,K-ATPase in collecting duct principal cells. - Wikidata - wikimedia - TriplyDB

Sodium transport is modulated by p38 kinase-dependent cross-talk between ENaC and Na,K-ATPase in collecting duct principal cells.

Sodium transport is modulated by p38 kinase-dependent cross-talk between ENaC and Na,K-ATPase in collecting duct principal cells.

http://www.wikidata.org/entity/Q37524110

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CPT-cGMP Is A New Ligand of Epithelial Sodium Channels A functional 12T-insertion polymorphism in the ATP1A1 promoter confers decreased susceptibility to hypertension in a male Sardinian population Paradoxical activation of the sodium chloride cotransporter (NCC) without hypertension in kidney deficient in a regulatory subunit of Na,K-ATPase, FXYD2.Transcriptional regulators of Na,K-ATPase subunits.Binding of FGF2 to FGFR2 in an autocrine mode in trophectoderm cells is indispensable for mouse blastocyst formation through PKC-p38 pathway Downregulation of Organic Anion Transporting Polypeptide (OATP) 1B1 Transport Function by Lysosomotropic Drug Chloroquine: Implication in OATP-Mediated Drug-Drug Interactions Urinary serine proteases and activation of ENaC in kidney--implications for physiological renal salt handling and hypertensive disorders with albuminuria.Coordinated Control of ENaC and Na+,K+-ATPase in Renal Collecting Duct.Dietary sodium induces a redistribution of the tubular metabolic workload.Effects of gonadectomy and testosterone treatment on aquaporin expression in the kidney of normotensive and hypertensive rats.Cell-specific diversity in the expression and organization of cytoplasmic plaque proteins of apical junctions.

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Sodium transport is modulated by p38 kinase-dependent cross-talk between ENaC and Na,K-ATPase in collecting duct principal cells.

http://www.wikidata.org/entity/Q37524110

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 31 October 2013

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Sodium transport is modulated ...... llecting duct principal cells.

@en

Sodium transport is modulated ...... llecting duct principal cells.

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type

label

Sodium transport is modulated ...... llecting duct principal cells.

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Sodium transport is modulated ...... llecting duct principal cells.

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prefLabel

Sodium transport is modulated ...... llecting duct principal cells.

@en

Sodium transport is modulated ...... llecting duct principal cells.

@nl

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Journal of the American Society of Nephrology

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Sodium transport is modulated ...... llecting duct principal cells.

@en

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Alain Doucet

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Arvid B Maunsbach

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Eric Féraille

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Eva Dizin

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Pierre-Yves Martin

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Sophie de Seigneux

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Thomas Ernandez

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Udo Hasler

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Valérie Leroy

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Yu-Bao Wang

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P2860

ATF-2 contains a phosphorylation-dependent transcriptional activation domain

α-Actinin-4/FSGS1 is required for Arp2/3-dependent actin assembly at the adherens junction

Aldosterone induces rapid apical translocation of ENaC in early portion of renal collecting system: possible role of SGK.

Inhibitors of protein biosynthesis. II. Mode of action of anisomycin.

AS160 associates with the Na+,K+-ATPase and mediates the adenosine monophosphate-stimulated protein kinase-dependent regulation of sodium pump surface expression

Sodium and potassium in the pathogenesis of hypertension.

Overexpression of the epithelial Na+ channel gamma subunit in collecting duct cells: interactions of Liddle's mutations and steroids on expression and function.

Activation of p38 mitogen-activated protein kinase promotes epidermal growth factor receptor internalization.

Cyclic AMP increases cell surface expression of functional Na,K-ATPase units in mammalian cortical collecting duct principal cells.

p38 MAP kinase mediates stress-induced internalization of EGFR: implications for cancer chemotherapy.

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250-259

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scholarly article

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10.1681/ASN.2013040429

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2

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25

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2013-10-31T00:00:00Z

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24179170

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3904566

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