Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity. - Wikidata - wikimedia - TriplyDB

Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

http://www.wikidata.org/entity/Q37524210

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The NF-κB Pathway and Cancer Stem Cells Cancer microenvironment, inflammation and cancer stem cells: A hypothesis for a paradigm change and new targets in cancer control Aberrant nuclear factor-kappa B activity in acute myeloid leukemia: from molecular pathogenesis to therapeutic target Update on acute myeloid leukemia stem cells: New discoveries and therapeutic opportunities Stress and Non-Stress Roles of Inflammatory Signals during HSC Emergence and Maintenance A Phase 2 study of bortezomib combined with either idarubicin/cytarabine or cytarabine/etoposide in children with relapsed, refractory or secondary acute myeloid leukemia: a report from the Children's Oncology Group A rationally designed nanoparticle for RNA interference therapy in B-lineage lymphoid malignancies Loss of IKKβ but Not NF-κB p65 Skews Differentiation towards Myeloid over Erythroid Commitment and Increases Myeloid Progenitor Self-Renewal and Functional Long-Term Hematopoietic Stem Cells.AMPK-Activated Protein Kinase Suppresses Ccr2 Expression by Inhibiting the NF-κB Pathway in RAW264.7 Macrophages Curcumin effectively inhibits oncogenic NF-κB signaling and restrains stemness features in liver cancer.Systematic Genome-wide Screening and Prediction of microRNAs in EBOV During the 2014 Ebolavirus Outbreak.EPA Prevents the Development of Abdominal Aortic Aneurysms through Gpr-120/Ffar-4.Correlation between p65 and TNF-α in patients with acute myelocytic leukemia.IL-32θ gene expression in acute myeloid leukemia suppresses TNF-α production.Transient potential receptor melastatin-2 (Trpm2) does not influence murine MLL-AF9-driven AML leukemogenesis or in vitro response to chemotherapy.A dual role for Caspase8 and NF-κB interactions in regulating apoptosis and necroptosis of ovarian cancer, with correlation to patient survival MLL-Rearranged Leukemias-An Update on Science and Clinical Approaches.Sensitizing leukemia stem cells to NF-κB inhibitor treatment in vivo by inactivation of both TNF and IL-1 signaling.Regulation of hematopoietic and leukemic stem cells by the immune system.Molecular targets of luteolin in cancer Biology and Clinical Relevance of Acute Myeloid Leukemia Stem Cells.Therapeutically targeting SELF-reinforcing leukemic niches in acute myeloid leukemia: A worthy endeavor?Chemopreventive Strategies for Inflammation-Related Carcinogenesis: Current Status and Future Direction.Let-7a-3 hypomethylation is associated with favorable/intermediate karyotypes but not with survival in acute myeloid leukemia.The role of the proteasome in AML.Leukemic stem cells: identification and clinical application.Genetic polymorphisms of IL-18 rs1946518 and IL-1β rs16944 are associated with prognosis and survival of acute myeloid leukemia.Mass cytometry analysis reveals hyperactive NF Kappa B signaling in myelofibrosis and secondary acute myeloid leukemia.Meta-Analysis of Gene Expression Profiles in Acute Promyelocytic Leukemia Reveals Involved Pathways.Inhibition of tumor necrosis factor-α enhances apoptosis induced by nuclear factor-κB inhibition in leukemia cells.Fanconi Anemia Mesenchymal Stromal Cells-Derived Glycerophospholipids Skew Hematopoietic Stem Cell Differentiation Through Toll-Like Receptor Signaling.Lippia origanoides extract induces cell cycle arrest and apoptosis and suppresses NF-κB signaling in triple-negative breast cancer cells.Inflammatory Signaling Pathways in Preleukemic and Leukemic Stem Cells.TNFα and TGFβ-1 synergistically increase the cancer stem cell properties of MiaPaCa-2 cells.MLL2, Not MLL1, Plays a Major Role in Sustaining MLL-Rearranged Acute Myeloid Leukemia.IL-32θ: a recently identified anti-inflammatory variant of IL-32 and its preventive role in various disorders and tumor suppressor activity.NR4A1 and NR4A3 restrict HSC proliferation via reciprocal regulation of C/EBPα and inflammatory signaling.Metastatic ability and the epithelial-mesenchymal transition in induced cancer stem-like hepatoma cells.Effective targeting of primitive AML CD34+ cells by the second-generation proteasome inhibitor carfilzomib.Effect of expressions of tumor necrosis factor α and interleukin 1B on peritoneal metastasis of gastric cancer.

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Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

http://www.wikidata.org/entity/Q37524210

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 02 January 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

@en

Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

@nl

type

label

Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

@en

Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

@nl

prefLabel

Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

@en

Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

@nl

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Journal of Clinical Investigation

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Positive feedback between NF-κB and TNF-α promotes leukemia-initiating cell capacity.

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Hiroshi Kobayashi

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Keiki Kumano

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Keisuke Kataoka

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Mineo Kurokawa

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Shunya Arai

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Taku Saito

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Yuki Kagoya

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P2860

Recurring mutations found by sequencing an acute myeloid leukemia genome

Human acute myeloid leukemia is organized as a hierarchy that originates from a primitive hematopoietic cell

A cell initiating human acute myeloid leukaemia after transplantation into SCID mice

Analyzing real-time PCR data by the comparative C(T) method

Nuclear factor kappaB-dependent gene expression profiling of Hodgkin's disease tumor cells, pathogenetic significance, and link to constitutive signal transducer and activator of transcription 5a activity

Differential regulation of the c-myc oncogene promoter by the NF-kappa B rel family of transcription factors

Tumor necrosis factor and interleukin-1 lead to phosphorylation and loss of I kappa B alpha: a mechanism for NF-kappa B activation

Nuclear factor-kappaB in cancer development and progression

NF-kappaB: linking inflammation and immunity to cancer development and progression

NF-kappaB functions as a tumour promoter in inflammation-associated cancer

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528-542

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scholarly article

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10.1172/JCI68101

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2

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124

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Akihide Yoshimi

Yoichiro Iwakura

Masahiro Nakagawa

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2014-01-02T00:00:00Z

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259559644

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24382349

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3904603

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