NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
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Alcoholic liver disease and hepatitis C: a frequently underestimated combinationBeverage specific alcohol intake in a population-based study: evidence for a positive association between pulmonary function and wine intakeMacrophages and Alcohol-Related Liver InflammationKupffer cells in the liverHypertension and chronic ethanol consumption: What do we know after a century of study?Chronic granulomatous diseaseTargeting the redox balance in inflammatory skin conditionsCritical Roles of Kupffer Cells in the Pathogenesis of Alcoholic Liver Disease: From Basic Science to Clinical TrialsOxidative stress, cardiolipin and mitochondrial dysfunction in nonalcoholic fatty liver diseaseOxidative stress and protein oxidation in the brain of water drinking and alcohol drinking rats administered the HIV envelope protein, gp120ERK1/2 and Egr-1 contribute to increased TNF-alpha production in rat Kupffer cells after chronic ethanol feedingSynergistic activation of glucose-6-phosphate dehydrogenase and NAD(P)H oxidase by Src kinase elevates superoxide in type 2 diabetic, Zucker fa/fa, rat liverEthanol-induced HO-1 and NQO1 are differentially regulated by HIF-1alpha and Nrf2 to attenuate inflammatory cytokine expressionLiver fibrosisIncreased oxidation and degradation of cytosolic proteins in alcohol-exposed mouse liver and hepatoma cells.Influence of chronic alcohol consumption on inward rectifier potassium channels in cerebral arteriolesResveratrol inhibits nonalcoholic fatty liver disease in ratsNADPH oxidase limits innate immune responses in the lungs in miceRole of NADPH oxidases in liver fibrosis.Maternal alcohol use during pregnancy causes systemic oxidation of the glutathione redox system.A simple method to systematically study oxidatively modified proteins in biological samples and its applicationsOxidative stress and hepatic Nox proteins in chronic hepatitis C and hepatocellular carcinoma.Toll-like receptor 4 and hepatic fibrogenesisCYP2E1 potentiation of LPS and TNFα-induced hepatotoxicity by mechanisms involving enhanced oxidative and nitrosative stress, activation of MAP kinases, and mitochondrial dysfunction.Nitric oxide and redox regulation in the liver: Part I. General considerations and redox biology in hepatitisEpigenetic Interactions between Alcohol and Cannabinergic Effects: Focus on Histone Modification and DNA Methylation.Exogenous thioredoxin prevents ethanol-induced oxidative damage and apoptosis in mouse liver.The presence of p47phox in liver parenchymal cells is a key mediator in the pathogenesis of alcoholic liver steatosisImpaired pulmonary NF-kappaB activation in response to lipopolysaccharide in NADPH oxidase-deficient mice.Mitochondria-targeted cytochrome P450 2E1 induces oxidative damage and augments alcohol-mediated oxidative stress.Toll-like receptors in the pathogenesis of alcoholic liver disease.A superoxide-hypersusceptible Salmonella enterica serovar Typhimurium mutant is attenuated but regains virulence in p47(phox-/-) mice.Screening for stress-resistance mutations in the mouseImmunohistochemical characterization of hepatic malondialdehyde and 4-hydroxynonenal modified proteins during early stages of ethanol-induced liver injury.Involvement of hepatic innate immunity in alcoholic liver disease.Toll-like receptor-mediated signaling cascade as a regulator of the inflammation network during alcoholic liver disease.Treatment of alcoholic hepatitis.Alcohol and mitochondria: a dysfunctional relationshipGenetic ablation of phagocytic NADPH oxidase in mice limits TNFα-induced inflammation in the lungs but not other tissues.Kupffer cells are depleted with HIV immunodeficiency and partially recovered with antiretroviral immune reconstitution.
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NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 2000
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
@en
NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
@nl
type
label
NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
@en
NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
@nl
prefLabel
NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
@en
NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
@nl
P2093
P2860
P356
P1476
NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.
@en
P2093
Bradford BU
Dikalova A
Holland SM
Kadiiska MB
P2860
P304
P356
10.1172/JCI9020
P407
P577
2000-10-01T00:00:00Z