Insufficient ER-stress response causes selective mouse cerebellar granule cell degeneration resembling that seen in congenital disorders of glycosylation.
about
Endoplasmic Reticulum Stress and Ethanol NeurotoxicityNeurological aspects of human glycosylation disordersNeurodegeneration and unfolded-protein response in mice expressing a membrane-tethered flexible tail of PrPEndoplasmic reticulum chaperone GRP78 is involved in autophagy activation induced by ischemic preconditioning in neural cells.DPAGT1-CDG: Functional analysis of disease-causing pathogenic mutations and role of endoplasmic reticulum stress.A nationwide survey of PMM2-CDG in Italy: high frequency of a mild neurological variant associated with the L32R mutation.
P2860
Insufficient ER-stress response causes selective mouse cerebellar granule cell degeneration resembling that seen in congenital disorders of glycosylation.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 04 December 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Insufficient ER-stress respons ...... al disorders of glycosylation.
@en
Insufficient ER-stress respons ...... al disorders of glycosylation.
@nl
type
label
Insufficient ER-stress respons ...... al disorders of glycosylation.
@en
Insufficient ER-stress respons ...... al disorders of glycosylation.
@nl
prefLabel
Insufficient ER-stress respons ...... al disorders of glycosylation.
@en
Insufficient ER-stress respons ...... al disorders of glycosylation.
@nl
P2093
P2860
P356
P1433
P1476
Insufficient ER-stress respons ...... al disorders of glycosylation.
@en
P2093
Liangwu Sun
Yingjun Zhao
Yun-Wu Zhang
P2860
P2888
P356
10.1186/1756-6606-6-52
P577
2013-12-04T00:00:00Z
P5875
P6179
1049909722