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Heregulin ameliorates the dystrophic phenotype in mdx mice.

Heregulin ameliorates the dystrophic phenotype in mdx mice.

http://www.wikidata.org/entity/Q37533952

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Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle Drug treatment of Duchenne muscular dystrophy: available evidence and perspectives IRES-mediated translation of utrophin A is enhanced by glucocorticoid treatment in skeletal muscle cells Drug discovery for Duchenne muscular dystrophy via utrophin promoter activation screening Daily treatment with SMTC1100, a novel small molecule utrophin upregulator, dramatically reduces the dystrophic symptoms in the mdx mouse Translational regulation of utrophin by miRNAs Ets-2 repressor factor silences extrasynaptic utrophin by N-box mediated repression in skeletal muscle.Functional substitution by TAT-utrophin in dystrophin-deficient mice.Dystrophic Cardiomyopathy-Potential Role of Calcium in Pathogenesis, Treatment and Novel Therapies Dystrophins, utrophins, and associated scaffolding complexes: role in mammalian brain and implications for therapeutic strategies.Serum Neuregulin-1beta as a Biomarker of Cardiovascular Fitness.A cell-based high-throughput screening assay for posttranscriptional utrophin upregulation.Biglycan recruits utrophin to the sarcolemma and counters dystrophic pathology in mdx mice High throughput screening in duchenne muscular dystrophy: from drug discovery to functional genomics Targeting artificial transcription factors to the utrophin A promoter: effects on dystrophic pathology and muscle function.Current status of pharmaceutical and genetic therapeutic approaches to treat DMD.PGC-1alpha regulates the neuromuscular junction program and ameliorates Duchenne muscular dystrophy.Neuregulin 1 Improves Glucose Tolerance in db/db Mice The muscular dystrophies: from genes to therapies.Viral-mediated gene therapy for the muscular dystrophies: successes, limitations and recent advances Overexpression of the cytotoxic T cell (CT) carbohydrate inhibits muscular dystrophy in the dyW mouse model of congenital muscular dystrophy 1A.Linker molecules between laminins and dystroglycan ameliorate laminin-alpha2-deficient muscular dystrophy at all disease stages Therapy insight: cardiovascular complications associated with muscular dystrophies.Emerging drugs for Duchenne muscular dystrophy Combinatorial therapeutic activation with heparin and AICAR stimulates additive effects on utrophin A expression in dystrophic muscles.Inhibition of muscle fibrosis results in increases in both utrophin levels and the number of revertant myofibers in Duchenne muscular dystrophy.Sarcospan: a small protein with large potential for Duchenne muscular dystrophy 2015 William Allan Award.Neuregulin in cardiovascular development and disease Overexpression of Galgt2 in skeletal muscle prevents injury resulting from eccentric contractions in both mdx and wild-type mice Overexpression of Galgt2 reduces dystrophic pathology in the skeletal muscles of alpha sarcoglycan-deficient mice The synaptic CT carbohydrate modulates binding and expression of extracellular matrix proteins in skeletal muscle: Partial dependence on utrophin.Identification of serum protein biomarkers for utrophin based DMD therapy.Progress in therapy for Duchenne muscular dystrophy.Cell-matrix interactions in muscle disease.Update on the treatment of Duchenne muscular dystrophy.Duchenne muscular dystrophy drug discovery - the application of utrophin promoter activation screening.What has the mdx mouse model of Duchenne muscular dystrophy contributed to our understanding of this disease?Advances in genetic therapeutic strategies for Duchenne muscular dystrophy.Overexpression of SERCA1a in the mdx diaphragm reduces susceptibility to contraction-induced damage.

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Heregulin ameliorates the dystrophic phenotype in mdx mice.

http://www.wikidata.org/entity/Q37533952

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article científic

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article scientifique

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artigo científico

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bilimsel makale

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scientific article published on 13 September 2004

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Heregulin ameliorates the dystrophic phenotype in mdx mice.

@en

Heregulin ameliorates the dystrophic phenotype in mdx mice.

@nl

type

label

Heregulin ameliorates the dystrophic phenotype in mdx mice.

@en

Heregulin ameliorates the dystrophic phenotype in mdx mice.

@nl

prefLabel

Heregulin ameliorates the dystrophic phenotype in mdx mice.

@en

Heregulin ameliorates the dystrophic phenotype in mdx mice.

@nl

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PNAS.0405972101

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Proceedings of the National Academy of Sciences of the United States of America

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Heregulin ameliorates the dystrophic phenotype in mdx mice.

@en

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Alan W Flake

http://www.w3.org/2001/XMLSchema#string

Claus J Jensen

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Elisabeth H Javazon

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Jacob Hansen-Schwartz

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M Dominik Fischer

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Sasha Bogdanovich

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Tejvir S Khurana

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Thomas O B Krag

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P2860

Utrophin-dystrophin-deficient mice as a model for Duchenne muscular dystrophy

Primary structure of dystrophin-related protein

An autosomal transcript in skeletal muscle with homology to dystrophin

Primary structure of dystrophin-associated glycoproteins linking dystrophin to the extracellular matrix

Association of dystrophin-related protein with dystrophin-associated proteins in mdx mouse muscle

Dystrophin: the protein product of the Duchenne muscular dystrophy locus

Complete cloning of the duchenne muscular dystrophy (DMD) cDNA and preliminary genomic organization of the DMD gene in normal and affected individuals

Systemic delivery of human microdystrophin to regenerating mouse dystrophic muscle by muscle progenitor cells

Understanding dystrophinopathies: an inventory of the structural and functional consequences of the absence of dystrophin in muscles of the mdx mouse.

Isolation of candidate cDNAs for portions of the Duchenne muscular dystrophy gene.

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13856-13860

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scholarly article

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10.1073/PNAS.0405972101

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