Fungal morphogenetic pathways are required for the hallmark inflammatory response during Candida albicans vaginitis. - Wikidata - wikimedia - TriplyDB

Fungal morphogenetic pathways are required for the hallmark inflammatory response during Candida albicans vaginitis.

Fungal morphogenetic pathways are required for the hallmark inflammatory response during Candida albicans vaginitis.

http://www.wikidata.org/entity/Q37546323

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Studies of Immune Responses in Candida vaginitis Mitochondrial Activity and Cyr1 Are Key Regulators of Ras1 Activation of C. albicans Virulence Pathways Phenotypic Profiling Reveals that Candida albicans Opaque Cells Represent a Metabolically Specialized Cell State Compared to Default White Cells Candida vaginitis: when opportunism knocks, the host responds Morphogenesis is not required for Candida albicans-Staphylococcus aureus intra-abdominal infection-mediated dissemination and lethal sepsis.Candida albicans cell-type switching and functional plasticity in the mammalian host.Microbial egress: a hitchhiker's guide to freedom.Secretory Aspartyl Proteinases Cause Vaginitis and Can Mediate Vaginitis Caused by Candida albicans in Mice.Transcriptomic analysis of vulvovaginal candidiasis identifies a role for the NLRP3 inflammasome.Ibuprofen potentiates the in vivo antifungal activity of fluconazole against Candida albicans murine infection Identification of novel mechanisms involved in generating localized vulvodynia pain.A Murine Model of Candida glabrata Vaginitis Shows No Evidence of an Inflammatory Immunopathogenic Response.Transcription Factors Efg1 and Bcr1 Regulate Biofilm Formation and Virulence during Candida albicans-Associated Denture Stomatitis ERG2 and ERG24 Are Required for Normal Vacuolar Physiology as Well as Candida albicans Pathogenicity in a Murine Model of Disseminated but Not Vaginal Candidiasis.Candida albicans VPS4 contributes differentially to epithelial and mucosal pathogenesis.Candida albicans-epithelial interactions and pathogenicity mechanisms: scratching the surface.Morphology-Independent Virulence of Candida Species during Polymicrobial Intra-abdominal Infections with Staphylococcus aureus.Association of a variable number tandem repeat in the NLRP3 gene in women with susceptibility to RVVC Global Role of Cyclic AMP Signaling in pH-Dependent Responses in Candida albicans.Vaginal Heparan Sulfate Linked to Neutrophil Dysfunction in the Acute Inflammatory Response Associated with Experimental Vulvovaginal Candidiasis.Vulvovaginal Candida albicans infections: pathogenesis, immunity and vaccine prospects.Vulvovaginal candidosis: contemporary challenges and the future of prophylactic and therapeutic approaches.Antimicrobial activity of Buchenavia tetraphylla against Candida albicans strains isolated from vaginal secretions.Contribution of TLR2 pathway in the pathogenesis of vulvovaginal candidiasis.Overexpression of Candida albicans Secreted Aspartyl Proteinases 2 or 5 is not sufficient for exacerbation of immunopathology in a murine model of vaginitis.Chemical inhibitors of Candida albicans hyphal morphogenesis target endocytosis.Control of mucosal candidiasis in the zebrafish swimbladder depends on neutrophils that block filament invasion and drive extracellular trap production.S. oralis activates the Efg1 filamentation pathway in C. albicans to promote cross-kingdom interactions and mucosal biofilms.The vaginal mycobiome: A contemporary perspective on fungi in women's health and diseases Antimicrobial activity, synergism and inhibition of germ tube formation by Crocus sativus-derived compounds against Candida spp.Inactivation of genes TEC1 and EFG1 in Candida albicans influences extracellular matrix composition and biofilm morphology Vocation, location, vocation: researching Candida pathogenesis.Candidalysin drives epithelial signaling, neutrophil recruitment, and immunopathology at the vaginal mucosa.A novel mechanism behind the immunopathogenesis of vulvovaginal candidiasis: "Neutrophil Anergy".Estrogen Receptor-Alpha (ESR1) Governs the Lower Female Reproductive Tract Vulnerability to Candida albicans.High-Throughput Screening Identifies Genes Required for Induction of Macrophage Pyroptosis The role of IL-33 in host response to Candida albicans

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Fungal morphogenetic pathways are required for the hallmark inflammatory response during Candida albicans vaginitis.

http://www.wikidata.org/entity/Q37546323

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年學術文章

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2013年學術文章

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Fungal morphogenetic pathways ...... ng Candida albicans vaginitis.

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Fungal morphogenetic pathways ...... ng Candida albicans vaginitis.

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Fungal morphogenetic pathways ...... ng Candida albicans vaginitis.

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Fungal morphogenetic pathways ...... ng Candida albicans vaginitis.

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Fungal morphogenetic pathways ...... ng Candida albicans vaginitis.

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Fungal morphogenetic pathways ...... ng Candida albicans vaginitis.

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Infection and Immunity

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Fungal morphogenetic pathways ...... ng Candida albicans vaginitis.

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Andrea K Nash

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Brian M Peters

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Elizabeth A Lilly

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Glen E Palmer

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Mairi C Noverr

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Paul L Fidel

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P2860

The danger model: a renewed sense of self

Evaluation of the role of Candida albicans agglutinin-like sequence (Als) proteins in human oral epithelial cell interactions

Nonfilamentous C. albicans mutants are avirulent

Regulation of cell-surface genes and biofilm formation by the C. albicans transcription factor Bcr1p.

Yeast transformation by the LiAc/SS Carrier DNA/PEG method.

Critical role of Bcr1-dependent adhesins in C. albicans biofilm formation in vitro and in vivo.

IL-1beta processing in host defense: beyond the inflammasomes.

Profile of Candida albicans-secreted aspartic proteinase elicited during vaginal infection.

Comparison between Candida albicans agglutinin-like sequence gene expression patterns in human clinical specimens and models of vaginal candidiasis.

Acute neutropenia decreases inflammation associated with murine vaginal candidiasis but has no effect on the course of infection.

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532-543

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10.1128/IAI.01417-13

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2

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