PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance
about
PPARs as determinants of the estrogen receptor lineage: use of synthetic lethality for the treatment of estrogen receptor-negative breast cancer.Transcriptomic response of breast cancer cells to anacardic acid.Pivotal Roles of Peroxisome Proliferator-Activated Receptors (PPARs) and Their Signal Cascade for Cellular and Whole-Body Energy Homeostasis.
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PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 18 December 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance
@en
PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance.
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type
label
PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance
@en
PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance.
@nl
prefLabel
PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance
@en
PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance.
@nl
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PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance
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P2093
Robert I Glazer
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P304
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10.1155/2016/3082340
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2016-12-18T00:00:00Z