Restoration of MYC-repressed targets mediates the negative effects of GM-CSF on RUNX1-ETO leukemogenicity. - Wikidata - wikimedia - TriplyDB

Restoration of MYC-repressed targets mediates the negative effects of GM-CSF on RUNX1-ETO leukemogenicity.

Restoration of MYC-repressed targets mediates the negative effects of GM-CSF on RUNX1-ETO leukemogenicity.

http://www.wikidata.org/entity/Q37559789

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c-Myc suppresses miR-451⊣YWTAZ/AKT axis via recruiting HDAC3 in acute myeloid leukemia.

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Restoration of MYC-repressed targets mediates the negative effects of GM-CSF on RUNX1-ETO leukemogenicity.

http://www.wikidata.org/entity/Q37559789

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 08 July 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Restoration of MYC-repressed t ...... on RUNX1-ETO leukemogenicity.

@en

Restoration of MYC-repressed t ...... on RUNX1-ETO leukemogenicity.

@nl

type

label

Restoration of MYC-repressed t ...... on RUNX1-ETO leukemogenicity.

@en

Restoration of MYC-repressed t ...... on RUNX1-ETO leukemogenicity.

@nl

prefLabel

Restoration of MYC-repressed t ...... on RUNX1-ETO leukemogenicity.

@en

Restoration of MYC-repressed t ...... on RUNX1-ETO leukemogenicity.

@nl

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Restoration of MYC-repressed t ...... on RUNX1-ETO leukemogenicity.

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A G Davis

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C T Mowery

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D Ran

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D-E Zhang

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K Lam

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S A Stoner

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S Weng

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Gadd45 stress sensors in malignancy and leukemia

An integrated database of genes responsive to the Myc oncogenic transcription factor: identification of direct genomic targets

Structural basis for recognition of SMRT/N-CoR by the MYND domain and its contribution to AML1/ETO's activity

Identification of breakpoints in t(8;21) acute myelogenous leukemia and isolation of a fusion transcript, AML1/ETO, with similarity to Drosophila segmentation gene, runt

Pretreatment cytogenetic abnormalities are predictive of induction success, cumulative incidence of relapse, and overall survival in adult patients with de novo acute myeloid leukemia: results from Cancer and Leukemia Group B (CALGB 8461)

Mxi1, a protein that specifically interacts with Max to bind Myc-Max recognition sites

Regulation of MYC expression and differential JQ1 sensitivity in cancer cells

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RNAi screen identifies Brd4 as a therapeutic target in acute myeloid leukaemia

MYC on the path to cancer

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159-169

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scholarly article

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10.1038/LEU.2016.167

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1

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Shinobu Matsuura

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2016-07-08T00:00:00Z

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