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Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

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Disruption of Steroid Axis, a New Paradigm for Molar Incisor Hypomineralization (MIH).Whole exome sequencing identifies an AMBN missense mutation causing severe autosomal-dominant amelogenesis imperfecta and dentin disorders

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Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

http://www.wikidata.org/entity/Q37565800

description

2016 թվականի հունվարին հրատարակված գիտական հոդված

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on January 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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name

Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

@en

Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

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type

label

Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

@en

Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

@nl

prefLabel

Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

@en

Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects.

@nl

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FPHYS.2016.00673

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Frontiers in Physiology

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Retinoic Acid Excess Impairs Amelogenesis Inducing Enamel Defects

@en

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Brigitte Schuhbaur

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Eric Mathieu

http://www.w3.org/2001/XMLSchema#string

Joseph Hemmerlé

http://www.w3.org/2001/XMLSchema#string

Karen Niederreither

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Pascal Dollé

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Supawich Morkmued

http://www.w3.org/2001/XMLSchema#string

Virginie Laugel-Haushalter

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P2860

Differential expression analysis for sequence count data

Craniosynostosis and multiple skeletal anomalies in humans and zebrafish result from a defect in the localized degradation of retinoic acid

Regulation of dental enamel shape and hardness

Homozygosity mapping and candidate prioritization identify mutations, missed by whole-exome sequencing, in SMOC2, causing major dental developmental defects

Amelogenin-deficient mice display an amelogenesis imperfecta phenotype

Enamelin (Enam) is essential for amelogenesis: ENU-induced mouse mutants as models for different clinical subtypes of human amelogenesis imperfecta (AI)

Enamelin is critical for ameloblast integrity and enamel ultrastructure formation

Estrogen and bisphenol A affect male rat enamel formation and promote ameloblast proliferation

Enamel defects and ameloblast-specific expression in Enam knock-out/lacz knock-in mice

Differential gene expression profiling of the molar tooth germ in peroxisome proliferator-activated receptor-alpha (PPAR-alpha) knockout mouse and in wild-type mouse: molar tooth phenotype of PPAR-alpha knockout mouse

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673

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scholarly article

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10.3389/FPHYS.2016.00673

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7

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