The main rhinovirus respiratory tract adhesion site (ICAM-1) is upregulated in smokers and patients with chronic airflow limitation (CAL).
about
Active smoking and COPD phenotype: distribution and impact on prognostic factors.Blocking rhinoviral adhesion molecule (ICAM-1): potential to prevent COPD exacerbations.Novel therapeutic approaches to simultaneously target rhinovirus infection and asthma/COPD pathogenesis.Pneumococcal vaccination and chronic respiratory diseases.PM2.5-induced oxidative stress increases intercellular adhesion molecule-1 expression in lung epithelial cells through the IL-6/AKT/STAT3/NF-κB-dependent pathway.
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The main rhinovirus respiratory tract adhesion site (ICAM-1) is upregulated in smokers and patients with chronic airflow limitation (CAL).
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 05 January 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The main rhinovirus respirator ...... onic airflow limitation (CAL).
@en
The main rhinovirus respiratory tract adhesion site
@nl
type
label
The main rhinovirus respirator ...... onic airflow limitation (CAL).
@en
The main rhinovirus respiratory tract adhesion site
@nl
prefLabel
The main rhinovirus respirator ...... onic airflow limitation (CAL).
@en
The main rhinovirus respiratory tract adhesion site
@nl
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The main rhinovirus respirator ...... onic airflow limitation (CAL).
@en
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Eugene Haydn Walters
Hans Konrad Muller
Malik Quasir Mahmood
Roger Latham
Steven Weston
Sukhwinder Singh Sohal
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P2888
P356
10.1186/S12931-016-0483-8
P577
2017-01-05T00:00:00Z
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1050419467