Therapy and clinical trials in frontotemporal dementia: past, present, and future. - Wikidata - wikimedia - TriplyDB

Therapy and clinical trials in frontotemporal dementia: past, present, and future.

Therapy and clinical trials in frontotemporal dementia: past, present, and future.

http://www.wikidata.org/entity/Q37566888

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Frontotemporal Dementia.Pharmacological Management of Psychiatric Symptoms in Frontotemporal Dementia: A Systematic Review.Updated meta-analysis of the role of APOE ε2/ε3/ε4 alleles in frontotemporal lobar degeneration.[Pathomechanisms and clinical aspects of frontotemporal lobar degeneration].Progranulin: a new avenue towards the understanding and treatment of neurodegenerative disease.Progranulin plasma levels predict the presence of GRN mutations in asymptomatic subjects and do not correlate with brain atrophy: results from the GENFI study.Frontotemporal dementia: latest evidence and clinical implications.TDP-43 as a potential biomarker for amyotrophic lateral sclerosis: a systematic review and meta-analysis.

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Therapy and clinical trials in frontotemporal dementia: past, present, and future.

http://www.wikidata.org/entity/Q37566888

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 15 June 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Therapy and clinical trials in frontotemporal dementia: past, present, and future.

@en

Therapy and clinical trials in frontotemporal dementia: past, present, and future.

@nl

type

label

Therapy and clinical trials in frontotemporal dementia: past, present, and future.

@en

Therapy and clinical trials in frontotemporal dementia: past, present, and future.

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prefLabel

Therapy and clinical trials in frontotemporal dementia: past, present, and future.

@en

Therapy and clinical trials in frontotemporal dementia: past, present, and future.

@nl

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Journal of Neurochemistry

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Therapy and clinical trials in frontotemporal dementia: past, present, and future.

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Richard M Tsai

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Modulation of the dynamic instability of tubulin assembly by the microtubule-associated protein tau

RNA toxicity from the ALS/FTD C9ORF72 expansion is mitigated by antisense intervention

Classification of primary progressive aphasia and its variants

Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia

Selective inhibition of Alzheimer disease-like tau aggregation by phenothiazines

Unconventional translation of C9ORF72 GGGGCC expansion generates insoluble polypeptides specific to c9FTD/ALS

Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS

A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD

Microtubule-stabilizing agents as potential therapeutics for neurodegenerative disease

Invited review: Frontotemporal dementia caused by microtubule-associated protein tau gene (MAPT) mutations: a chameleon for neuropathology and neuroimaging

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211-221

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scholarly article

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10.1111/JNC.13640

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138 Suppl 1

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2016-06-15T00:00:00Z

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27306957

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