Persistent infection with primate foamy virus type 1 increases human immunodeficiency virus type 1 cell binding via a Bet-independent mechanism. - Wikidata - wikimedia - TriplyDB

Persistent infection with primate foamy virus type 1 increases human immunodeficiency virus type 1 cell binding via a Bet-independent mechanism.

Persistent infection with primate foamy virus type 1 increases human immunodeficiency virus type 1 cell binding via a Bet-independent mechanism.

http://www.wikidata.org/entity/Q37568073

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Restriction of foamy viruses by APOBEC cytidine deaminases Dual Simian Foamy Virus/Human Immunodeficiency Virus Type 1 Infections in Persons from Côte d'Ivoire.Centrosomal latency of incoming foamy viruses in resting cells.Expanded tissue targets for foamy virus replication with simian immunodeficiency virus-induced immunosuppression.In vivo cellular tropism of gorilla simian foamy virus in blood of infected humans Foamy virus infection in primates.Influence of naturally occurring simian foamy viruses (SFVs) on SIV disease progression in the rhesus macaque (Macaca mulatta) model.Innate sensing of foamy viruses by human hematopoietic cells.

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P2860

Persistent infection with primate foamy virus type 1 increases human immunodeficiency virus type 1 cell binding via a Bet-independent mechanism.

http://www.wikidata.org/entity/Q37568073

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on October 2004

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Persistent infection with prim ...... a a Bet-independent mechanism.

@en

Persistent infection with prim ...... a a Bet-independent mechanism.

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type

label

Persistent infection with prim ...... a a Bet-independent mechanism.

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Persistent infection with prim ...... a a Bet-independent mechanism.

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prefLabel

Persistent infection with prim ...... a a Bet-independent mechanism.

@en

Persistent infection with prim ...... a a Bet-independent mechanism.

@nl

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P356

JVI.78.20.11405-11410.2004

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Journal of Virology

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Persistent infection with prim ...... a a Bet-independent mechanism.

@en

P2093

Abdelkrim Mannioui

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Ali Saib

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Bruno Canque

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Cecile Schiffer

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Charles-Henri Lecellier

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Elisabeth Nelson

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Jean Claude Gluckman

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Marie-Louise Giron

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Nathalie Felix

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P2860

Down-regulation of CXCR4 by human herpesvirus 6 (HHV-6) and HHV-7

DC-SIGN, a dendritic cell-specific HIV-1-binding protein that enhances trans-infection of T cells

Detection, isolation, and continuous production of cytopathic retroviruses (HTLV-III) from patients with AIDS and pre-AIDS

Functions of cell surface heparan sulfate proteoglycans

Foamy viruses are unconventional retroviruses

Human immunodeficiency virus type 1 attachment to HeLa CD4 cells is CD4 independent and gp120 dependent and requires cell surface heparans.

Foamy viruses: between retroviruses and pararetroviruses.

HHV-6 infection in patients with HIV-1 infection and disease.

Mediation of human immunodeficiency virus type 1 binding by interaction of cell surface heparan sulfate proteoglycans with the V3 region of envelope gp120-gp41.

Involvement of a spliced and defective human foamy virus in the establishment of chronic infection.

P304

11405-11410

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scholarly article

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10.1128/JVI.78.20.11405-11410.2004

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Jacqueline Lehmann-Che

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2004-10-01T00:00:00Z

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15452263

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