Susceptibility of podocytes to palmitic acid is regulated by fatty acid oxidation and inversely depends on acetyl-CoA carboxylases 1 and 2. - Wikidata - wikimedia - TriplyDB

Susceptibility of podocytes to palmitic acid is regulated by fatty acid oxidation and inversely depends on acetyl-CoA carboxylases 1 and 2.

Susceptibility of podocytes to palmitic acid is regulated by fatty acid oxidation and inversely depends on acetyl-CoA carboxylases 1 and 2.

http://www.wikidata.org/entity/Q37575477

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Podocytes Obesity-Related Chronic Kidney Disease-The Role of Lipid Metabolism Free Fatty acids and their metabolism affect function and survival of podocytes Interaction of thrombospondin1 and CD36 contributes to obesity-associated podocytopathy.Fetuin-A aggravates lipotoxicity in podocytes via interleukin-1 signaling.Lipotoxicity as a trigger factor of renal disease.Dyslipidaemia in nephrotic syndrome: mechanisms and treatment.Overexpression of heart-type fatty acid binding protein enhances fatty acid-induced podocyte injury.Impaired β-Oxidation and Altered Complex Lipid Fatty Acid Partitioning with Advancing CKD.Identification of common and differential mechanisms of glomerulus and tubule senescence in 24-month-old rats by quantitative LC-MS/MS.Lipid peroxidation regulates podocyte migration and cytoskeletal structure through redox sensitive RhoA signaling.

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Susceptibility of podocytes to palmitic acid is regulated by fatty acid oxidation and inversely depends on acetyl-CoA carboxylases 1 and 2.

http://www.wikidata.org/entity/Q37575477

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article científic

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article scientifique

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bilimsel makale

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scientific article published on 11 December 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Susceptibility of podocytes to ...... etyl-CoA carboxylases 1 and 2.

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AJPRENAL.00454.2013

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American Journal of Physiology - Renal Physiology

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Susceptibility of podocytes to ...... etyl-CoA carboxylases 1 and 2.

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Andreas Werner Jehle

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Jana Marina Orellana

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Kapil Kampe

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Peter Mundel

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P2860

Oral Pharmacologic Treatment of Type 2 Diabetes Mellitus: A Clinical Practice Guideline From the American College of Physicians

Adiponectin regulates albuminuria and podocyte function in mice

Mutant mice lacking acetyl-CoA carboxylase 1 are embryonically lethal

Gene knockout of Acc2 has little effect on body weight, fat mass, or food intake

Endoplasmic reticulum stress in the β-cell pathogenesis of type 2 diabetes

The glucose fatty-acid cycle. Its role in insulin sensitivity and the metabolic disturbances of diabetes mellitus

AMPK: a nutrient and energy sensor that maintains energy homeostasis

A single nucleotide polymorphism within the acetyl-coenzyme A carboxylase beta gene is associated with proteinuria in patients with type 2 diabetes.

Uptake of individual free fatty acids by skeletal muscle and liver in man.

Regulation of podocyte survival and endoplasmic reticulum stress by fatty acids

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