Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia. - Wikidata - wikimedia - TriplyDB

Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.

Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.

http://www.wikidata.org/entity/Q37576073

about

Pathogenesis of bronchopulmonary dysplasia: when inflammation meets organ development The Extracellular Matrix in Bronchopulmonary Dysplasia: Target and Source Cannabidiol, a Major Non-Psychotropic Cannabis Constituent Enhances Fracture Healing and Stimulates Lysyl Hydroxylase Activity in Osteoblasts.Animal models of bronchopulmonary dysplasia. The preterm baboon models.Animal models of bronchopulmonary dysplasia. The term mouse models Conditional overexpression of TGFβ1 promotes pulmonary inflammation, apoptosis and mortality via TGFβR2 in the developing mouse lung.Lung matrix and vascular remodeling in mechanically ventilated elastin haploinsufficient newborn mice.The H2S-generating enzymes cystathionine β-synthase and cystathionine γ-lyase play a role in vascular development during normal lung alveolarization.Cyclophilin-B Modulates Collagen Cross-linking by Differentially Affecting Lysine Hydroxylation in the Helical and Telopeptidyl Domains of Tendon Type I Collagen.Coming to terms with tissue engineering and regenerative medicine in the lung.The roles of Tenascin C and Fibronectin 1 in adhesive capsulitis: a pilot gene expression study.Advances in paediatric pulmonary vascular disease associated with bronchopulmonary dysplasia.LungMAP: The Molecular Atlas of Lung Development Program.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Looking ahead: where to next for animal models of bronchopulmonary dysplasia?Collagen and elastin cross-linking is altered during aberrant late lung development associated with hyperoxia.Searching for better animal models of BPD: a perspective.Transforming growth factor-β stimulates Smad1/5 signaling in pulmonary artery smooth muscle cells and fibroblasts of the newborn mouse through ALK1.Expanding the Clinical Spectrum of Phenotypes Caused by Pathogenic Variants in PLOD2.Extracellular matrix in lung development, homeostasis and disease.Platelet-rich plasma for the treatment of patent ductus arteriosus: not quite ready for prime time.The Potentials and Caveats of Mesenchymal Stromal Cell-Based Therapies in the Preterm Infant.Overexpression of PLOD3 promotes tumor progression and poor prognosis in gliomas.

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P2860

Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.

http://www.wikidata.org/entity/Q37576073

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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2013年學術文章

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name

Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.

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Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.

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Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.

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Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.

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Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.

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Elpidoforos Sakkas

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Gero Niess

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Jesse D Roberts

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Konstantin Mayer

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Pawel Turowski

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Simone Becker

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Thilo J Witsch

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P2860

Bronchopulmonary dysplasia

Pulmonary disease following respirator therapy of hyaline-membrane disease. Bronchopulmonary dysplasia

Glycosylation catalyzed by lysyl hydroxylase 3 is essential for basement membranes

Lysyl oxidase activity is dysregulated during impaired alveolarization of mouse and human lungs.

Mutations in the lysyl hydroxylase 1 gene that result in enzyme deficiency and the clinical phenotype of Ehlers-Danlos syndrome type VI.

Fox-2 protein regulates the alternative splicing of scleroderma-associated lysyl hydroxylase 2 messenger RNA

Lysyl hydroxylase 3 (LH3) modifies proteins in the extracellular space, a novel mechanism for matrix remodeling.

Identification of PLOD2 as telopeptide lysyl hydroxylase, an important enzyme in fibrosis.

Inhibiting lung elastase activity enables lung growth in mechanically ventilated newborn mice.

Curcumin augments lung maturation, preventing neonatal lung injury by inhibiting TGF-β signaling.

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