Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.
about
Pathogenesis of bronchopulmonary dysplasia: when inflammation meets organ developmentThe Extracellular Matrix in Bronchopulmonary Dysplasia: Target and SourceCannabidiol, a Major Non-Psychotropic Cannabis Constituent Enhances Fracture Healing and Stimulates Lysyl Hydroxylase Activity in Osteoblasts.Animal models of bronchopulmonary dysplasia. The preterm baboon models.Animal models of bronchopulmonary dysplasia. The term mouse modelsConditional overexpression of TGFβ1 promotes pulmonary inflammation, apoptosis and mortality via TGFβR2 in the developing mouse lung.Lung matrix and vascular remodeling in mechanically ventilated elastin haploinsufficient newborn mice.The H2S-generating enzymes cystathionine β-synthase and cystathionine γ-lyase play a role in vascular development during normal lung alveolarization.Cyclophilin-B Modulates Collagen Cross-linking by Differentially Affecting Lysine Hydroxylation in the Helical and Telopeptidyl Domains of Tendon Type I Collagen.Coming to terms with tissue engineering and regenerative medicine in the lung.The roles of Tenascin C and Fibronectin 1 in adhesive capsulitis: a pilot gene expression study.Advances in paediatric pulmonary vascular disease associated with bronchopulmonary dysplasia.LungMAP: The Molecular Atlas of Lung Development Program.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Looking ahead: where to next for animal models of bronchopulmonary dysplasia?Collagen and elastin cross-linking is altered during aberrant late lung development associated with hyperoxia.Searching for better animal models of BPD: a perspective.Transforming growth factor-β stimulates Smad1/5 signaling in pulmonary artery smooth muscle cells and fibroblasts of the newborn mouse through ALK1.Expanding the Clinical Spectrum of Phenotypes Caused by Pathogenic Variants in PLOD2.Extracellular matrix in lung development, homeostasis and disease.Platelet-rich plasma for the treatment of patent ductus arteriosus: not quite ready for prime time.The Potentials and Caveats of Mesenchymal Stromal Cell-Based Therapies in the Preterm Infant.Overexpression of PLOD3 promotes tumor progression and poor prognosis in gliomas.
P2860
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P2860
Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
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2013年學術文章
@zh-hant
name
Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.
@en
Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.
@nl
type
label
Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.
@en
Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.
@nl
prefLabel
Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.
@en
Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.
@nl
P2093
P2860
P50
P1476
Deregulation of the lysyl hydr ...... al bronchopulmonary dysplasia.
@en
P2093
Elpidoforos Sakkas
Gero Niess
Jesse D Roberts
Konstantin Mayer
Pawel Turowski
Simone Becker
Thilo J Witsch
P2860
P304
P356
10.1152/AJPLUNG.00109.2013
P577
2013-11-27T00:00:00Z