Viral evolution and interferon resistance of hepatitis C virus RNA replication in a cell culture model. - Wikidata - wikimedia - TriplyDB

Viral evolution and interferon resistance of hepatitis C virus RNA replication in a cell culture model.

Viral evolution and interferon resistance of hepatitis C virus RNA replication in a cell culture model.

http://www.wikidata.org/entity/Q37583537

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Hepatitis C virus protease NS3/4A cleaves mitochondrial antiviral signaling protein off the mitochondria to evade innate immunity Optineurin negatively regulates the induction of IFNbeta in response to RNA virus infection Regulating intracellular antiviral defense and permissiveness to hepatitis C virus RNA replication through a cellular RNA helicase, RIG-I The role of enterovirus in chronic fatigue syndrome Inhibition of interferon-stimulated JAK-STAT signaling by a tick-borne flavivirus and identification of NS5 as an interferon antagonist Replication of Hepatitis C Virus (HCV) RNA in Mouse Embryonic Fibroblasts: Protein Kinase R (PKR)-Dependent and PKR-Independent Mechanisms for Controlling HCV RNA Replication and Mediating Interferon Activities Novel Robust Hepatitis C Virus Mouse Efficacy Model Viral Determinants of Resistance to Treatment in Patients with Hepatitis C Evolution of Resistant M414T Mutants among Hepatitis C Virus Replicon Cells Treated with Polymerase Inhibitor A-782759 Characterization of Hepatitis C Virus Subgenomic Replicon Resistance to Cyclosporine In Vitro The C Terminus of Hepatitis C Virus NS4A Encodes an Electrostatic Switch That Regulates NS5A Hyperphosphorylation and Viral Replication Hepatitis C Virus Stabilizes Hypoxia-Inducible Factor 1 and Stimulates the Synthesis of Vascular Endothelial Growth Factor Reduced expression of Jak-1 and Tyk-2 proteins leads to interferon resistance in Hepatitis C virus replicon Blockade of virus infection by human CD4+ T cells via a cytokine relay network Preclinical Characteristics of the Hepatitis C Virus NS3/4A Protease Inhibitor ITMN-191 (R7227)Using high-throughput genomics to study hepatitis C: What determines the outcome of infection?Hepatitis C virus controls interferon production through PKR activation Proteomic analysis of mitochondrial-associated ER membranes (MAM) during RNA virus infection reveals dynamic changes in protein and organelle trafficking Convergent evolution of escape from hepaciviral antagonism in primates Control of antiviral defenses through hepatitis C virus disruption of retinoic acid-inducible gene-I signaling ENCODE tiling array analysis identifies differentially expressed annotated and novel 5' capped RNAs in hepatitis C infected liver.Antiviral potency analysis and functional comparison of consensus interferon, interferon-alpha2a and pegylated interferon-alpha2b against hepatitis C virus infection.Hepatitis C virus blocks interferon effector function by inducing protein kinase R phosphorylation.Ethanol and reactive species increase basal sequence heterogeneity of hepatitis C virus and produce variants with reduced susceptibility to antivirals.Identification and characterization of a ross river virus variant that grows persistently in macrophages, shows altered disease kinetics in a mouse model, and exhibits resistance to type I interferon The membrane-bound transcription factor CREB3L1 is activated in response to virus infection to inhibit proliferation of virus-infected cells.Epigenetic silencing of antiviral genes renders clones of Huh-7 cells permissive for hepatitis C virus replication Hepatitis C virus (HCV) employs multiple strategies to subvert the host innate antiviral response.Innate immune tolerance and the role of kupffer cells in differential responses to interferon therapy among patients with HCV genotype 1 infection.Identification of alpha interferon-induced envelope mutations of hepatitis C virus in vitro associated with increased viral fitness and interferon resistance.The broad-spectrum antiviral functions of IFIT and IFITM proteins.IFIT1: A dual sensor and effector molecule that detects non-2'-O methylated viral RNA and inhibits its translation.Quantitative microspectroscopic imaging reveals viral and cellular RNA helicase interactions in live cells.Tricistronic hepatitis C virus subgenomic replicon expressing double transgenes.Control of innate immune signaling and membrane targeting by the Hepatitis C virus NS3/4A protease are governed by the NS3 helix α0.Functional and therapeutic analysis of hepatitis C virus NS3.4A protease control of antiviral immune defense.Osteopontin regulates HCV replication and assembly via interaction with HCV proteins and lipid droplets and by binding to receptors, αVβ3 and CD44.Evolutionary Analysis Provides Insight Into the Origin and Adaptation of HCV.

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Viral evolution and interferon resistance of hepatitis C virus RNA replication in a cell culture model.

http://www.wikidata.org/entity/Q37583537

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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Viral evolution and interferon ...... ation in a cell culture model.

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Viral evolution and interferon ...... ation in a cell culture model.

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JVI.78.21.11591-11604.2004

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Viral evolution and interferon ...... ation in a cell culture model.

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Eileen Foy

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Rhea Sumpter

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Yueh-Ming Loo

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P2860

Inhibition of the interferon-inducible protein kinase PKR by HCV E2 protein

Alpha interferon induces distinct translational control programs to suppress hepatitis C virus RNA replication

A new pathway of translational regulation mediated by eukaryotic initiation factor 3

Antiapoptotic and oncogenic potentials of hepatitis C virus are linked to interferon resistance by viral repression of the PKR protein kinase

Expression of hepatitis C virus proteins inhibits signal transduction through the Jak-STAT pathway

DNA microarray analysis of chimpanzee liver during acute resolving hepatitis C virus infection.

Effect of alpha interferon on the hepatitis C virus replicon

Regulation of PKR and IRF-1 during hepatitis C virus RNA replication.

Nonlinear partial differential equations and applications: Genomic analysis of the host response to hepatitis C virus infection

Antiviral effect and virus-host interactions in response to alpha interferon, gamma interferon, poly(i)-poly(c), tumor necrosis factor alpha, and ribavirin in hepatitis C virus subgenomic replicons.

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