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Genetic testing in the management of inherited arrhythmia syndromes.

Genetic testing in the management of inherited arrhythmia syndromes.

http://www.wikidata.org/entity/Q37587475

about

Genetic testing for long QT syndrome and the category of cardiac ion channelopathies.Patient Recall, Interpretation, and Perspective of an Inconclusive Long QT Syndrome Genetic Test Result.

P2860

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P2860

Genetic testing in the management of inherited arrhythmia syndromes.

http://www.wikidata.org/entity/Q37587475

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on September 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Genetic testing in the management of inherited arrhythmia syndromes.

@en

Genetic testing in the management of inherited arrhythmia syndromes.

@nl

type

label

Genetic testing in the management of inherited arrhythmia syndromes.

@en

Genetic testing in the management of inherited arrhythmia syndromes.

@nl

prefLabel

Genetic testing in the management of inherited arrhythmia syndromes.

@en

Genetic testing in the management of inherited arrhythmia syndromes.

@nl

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S11886-009-0048-2

P1433

Current cardiology reports

P1476

Genetic testing in the management of inherited arrhythmia syndromes.

@en

P2093

Edward P Gerstenfeld

http://www.w3.org/2001/XMLSchema#string

Wendy S Tzou

http://www.w3.org/2001/XMLSchema#string

P2860

Mutations of the cardiac ryanodine receptor (RyR2) gene in familial polymorphic ventricular tachycardia

Spectrum of mutations in long-QT syndrome genes. KVLQT1, HERG, SCN5A, KCNE1, and KCNE2

Mutations in the cardiac ryanodine receptor gene (hRyR2) underlie catecholaminergic polymorphic ventricular tachycardia

Genotype-phenotype correlation in the long-QT syndrome: gene-specific triggers for life-threatening arrhythmias.

Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate. Implications for gene-specific therapy.

P2888

s11886-009-0048-2

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343-351

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scholarly article

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10.1007/S11886-009-0048-2

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5

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11

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2009-09-01T00:00:00Z

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19709494

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