Antigen-independent memory CD8 T cells do not develop during chronic viral infection. - Wikidata - wikimedia - TriplyDB

Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

http://www.wikidata.org/entity/Q37591855

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Analysis of CD127 and KLRG1 expression on hepatitis C virus-specific CD8+ T cells reveals the existence of different memory T-cell subsets in the peripheral blood and liver Molecular and cellular insights into T cell exhaustion Tissue-resident memory T cells Overcoming T cell exhaustion in infection and cancer T cell memory in the context of persistent herpes viral infections Liver-Infiltrating Lymphocytes in Chronic Human Hepatitis C Virus Infection Display an Exhausted Phenotype with High Levels of PD-1 and Low Levels of CD127 Expression Viral Vaccines and CTL Response The PD1:PD-L1/2 Pathway from Discovery to Clinical Implementation Innate and Adaptive Immune Regulation During Chronic Viral Infections Blimp-1 homolog Hobit identifies effector-type lymphocytes in humans Host-reactive CD8+ memory stem cells in graft-versus-host disease Restoring function in exhausted CD8 T cells during chronic viral infection Effector memory Th1 CD4 T cells are maintained in a mouse model of chronic malaria Lysine auxotrophy combined with deletion of the SecA2 gene results in a safe and highly immunogenic candidate live attenuated vaccine for tuberculosis Antigen load and viral sequence diversification determine the functional profile of HIV-1-specific CD8+ T cells HLA Class I-T cell epitopes from trans-sialidase proteins reveal functionally distinct subsets of CD8+ T cells in chronic Chagas disease.Changes in Trypanosoma cruzi-specific immune responses after treatment: surrogate markers of treatment efficacy.Delayed expansion and contraction of CD8+ T cell response during infection with virulent Salmonella typhimurium.De novo recruitment of antigen-experienced and naive T cells contributes to the long-term maintenance of antiviral T cell populations in the persistently infected central nervous system Induction of protective immunity against murine gammaherpesvirus 68 infection in the absence of viral latency.Memory CD8+ T cell differentiation Persistent expression of FLAG-tagged micro dystrophin in nonhuman primates following intramuscular and vascular delivery.Cutting edge: IL-7-independent regulation of IL-7 receptor alpha expression and memory CD8 T cell development.Shaping successful and unsuccessful CD8 T cell responses following infection.Aberrant plasma IL-7 and soluble IL-7 receptor levels indicate impaired T-cell response to IL-7 in human tuberculosis.Dynamics of viral evolution and CTL responses in HIV-1 infection Epigenetic stability of exhausted T cells limits durability of reinvigoration by PD-1 blockade.Limited transplantation of antigen-expressing hematopoietic stem cells induces long-lasting cytotoxic T cell responses Low CD8 T-cell proliferative potential and high viral load limit the effectiveness of therapeutic vaccination.Augmented replicative capacity of the boosting antigen improves the protective efficacy of heterologous prime-boost vaccine regimens.Altered T cell memory and effector cell development in chronic lymphatic filarial infection that is independent of persistent parasite antigen.Intrinsic functional dysregulation of CD4 T cells occurs rapidly following persistent viral infection Features of responding T cells in cancer and chronic infection.Identification of stem cell transcriptional programs normally expressed in embryonic and neural stem cells in alloreactive CD8+ T cells mediating graft-versus-host disease Exhausted CD8 T cells downregulate the IL-18 receptor and become unresponsive to inflammatory cytokines and bacterial co-infections.Non-hematopoietic cells in lymph nodes drive memory CD8 T cell inflation during murine cytomegalovirus infection.Chronic antigen stimulation alone is sufficient to drive CD8+ T cell exhaustion Tumor antigen-specific CD8 T cells infiltrating the tumor express high levels of PD-1 and are functionally impaired T-cell exhaustion: characteristics, causes and conversion.Pronounced virus-dependent activation drives exhaustion but sustains IFN-γ transcript levels.

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Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

http://www.wikidata.org/entity/Q37591855

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 25 October 2004

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

@en

Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

@nl

type

label

Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

@en

Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

@nl

prefLabel

Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

@en

Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

@nl

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PNAS.0407192101

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Proceedings of the National Academy of Sciences of the United States of America

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Antigen-independent memory CD8 T cells do not develop during chronic viral infection.

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Daniel L Barber

http://www.w3.org/2001/XMLSchema#string

E John Wherry

http://www.w3.org/2001/XMLSchema#string

Joseph N Blattman

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Rafi Ahmed

http://www.w3.org/2001/XMLSchema#string

Susan M Kaech

http://www.w3.org/2001/XMLSchema#string

P2860

Viral immune evasion due to persistence of activated T cells without effector function

Virus-Specific CD8+ Lymphocytes Share the Same Effector-Memory Phenotype but Exhibit Functional Differences in Acute Hepatitis B and C

Population biology of lymphocytes: the flight for survival

Lineage relationship and protective immunity of memory CD8 T cell subsets

IL-15Ralpha recycles and presents IL-15 In trans to neighboring cells

Expression of CD57 defines replicative senescence and antigen-induced apoptotic death of CD8+ T cells

Decay kinetics of human immunodeficiency virus-specific CD8+ T cells in peripheral blood after initiation of highly active antiretroviral therapy.

Interleukin-7 mediates the homeostasis of naïve and memory CD8 T cells in vivo.

Cytokine requirements for acute and Basal homeostatic proliferation of naive and memory CD8+ T cells.

CD4+CD25+ regulatory T cells control Leishmania major persistence and immunity.

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10.1073/PNAS.0407192101

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15505208

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524220

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