JNK regulates the release of proapoptotic mitochondrial factors in reovirus-infected cells. - Wikidata - wikimedia - TriplyDB

JNK regulates the release of proapoptotic mitochondrial factors in reovirus-infected cells.

JNK regulates the release of proapoptotic mitochondrial factors in reovirus-infected cells.

http://www.wikidata.org/entity/Q37596428

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Novel Strategy for Treatment of Viral Central Nervous System Infection by Using a Cell-Permeating Inhibitor of c-Jun N-Terminal Kinase Apoptosis in animal models of virus-induced disease Epizootic hemorrhagic disease virus induces and benefits from cell stress, autophagy, and apoptosis.Bid regulates the pathogenesis of neurotropic reovirus.Gene expression in the brain during reovirus encephalitis.Targeting cancer-initiating cells with oncolytic viruses.Oncolytic reovirus in canine mast cell tumor.Rotavirus activates JNK and p38 signaling pathways in intestinal cells, leading to AP-1-driven transcriptional responses and enhanced virus replication Uses for JNK: the many and varied substrates of the c-Jun N-terminal kinases.The oncolytic effects of reovirus in canine solid tumor cell lines.Posttranslational modification of vesicular stomatitis virus glycoprotein, but not JNK inhibition, is the antiviral mechanism of SP600125 Mechanisms of apoptosis during reovirus infection.Mechanisms of reovirus-induced cell death and tissue injury: role of apoptosis and virus-induced perturbation of host-cell signaling and transcription factor activation.Amplification of JNK signaling is necessary to complete the murine gammaherpesvirus 68 lytic replication cycle.Reovirus receptors, cell entry, and proapoptotic signaling.JNK1 Derived from Orange-Spotted Grouper, Epinephelus coioides, Involving in the Evasion and Infection of Singapore Grouper Iridovirus (SGIV).Daxx upregulation within the cytoplasm of reovirus-infected cells is mediated by interferon and contributes to apoptosis.Inhibition of NF-kappa B activity and cFLIP expression contribute to viral-induced apoptosis.Reovirus activates a caspase-independent cell death pathway.JNK-deficiency enhanced oncolytic vaccinia virus replication and blocked activation of double-stranded RNA-dependent protein kinase.Reovirus activates transforming growth factor beta and bone morphogenetic protein signaling pathways in the central nervous system that contribute to neuronal survival following infection.Fas-mediated apoptotic signaling in the mouse brain following reovirus infection Reovirus-based therapy for cancer.Activated ras signaling pathways and reovirus oncolysis: an update on the mechanism of preferential reovirus replication in cancer cells.The effects of oncolytic reovirus in canine lymphoma cell lines.JWA sensitizes P-glycoprotein-mediated drug-resistant choriocarcinoma cells to etoposide via JNK and mitochondrial-associated signal pathway.Poliovirus induces Bax-dependent cell death mediated by c-Jun NH2-terminal kinase.Bax is activated during rotavirus-induced apoptosis through the mitochondrial pathway.Oncolytic reovirus induces intracellular redistribution of Ras to promote apoptosis and progeny virus release.Viral gene expression potentiates reovirus-induced necrosis.REO-001: A phase I trial of percutaneous intralesional administration of reovirus type 3 dearing (Reolysin®) in patients with advanced solid tumors.Genes Induced by Reovirus Infection Have a Distinct Modular Cis-Regulatory Architecture Reovirus infection of the CNS enhances iNOS expression in areas of virus-induced injury.Reovirus therapy stimulates endoplasmic reticular stress, NOXA induction, and augments bortezomib-mediated apoptosis in multiple myeloma.Review: Oncolytic virotherapy, updates and future directions.Remifentanil preconditioning alleviating brain damage of cerebral ischemia reperfusion rats by regulating the JNK signal pathway and TNF-α/TNFR1 signal pathway.Going (Reo)Viral: Factors Promoting Successful Reoviral Oncolytic Infection

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JNK regulates the release of proapoptotic mitochondrial factors in reovirus-infected cells.

http://www.wikidata.org/entity/Q37596428

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scientific article published on December 2004

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JNK regulates the release of p ...... rs in reovirus-infected cells.

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JNK regulates the release of p ...... rs in reovirus-infected cells.

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JNK regulates the release of p ...... rs in reovirus-infected cells.

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JNK regulates the release of p ...... rs in reovirus-infected cells.

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JNK regulates the release of p ...... rs in reovirus-infected cells.

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JVI.78.23.13132-13138.2004

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Journal of Virology

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JNK regulates the release of p ...... ors in reovirus-infected cells

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Gary L Johnson

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Lisa A Moffitt

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Penny Clarke

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Sarah M Richardson-Burns

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Suzanne M Meintzer

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P2860

The HIV-1 viral protein R induces apoptosis via a direct effect on the mitochondrial permeability transition pore

JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

A protein kinase involved in the regulation of inflammatory cytokine biosynthesis

The Bax subfamily of Bcl2-related proteins is essential for apoptotic signal transduction by c-Jun NH(2)-terminal kinase

JNK phosphorylation of Bim-related members of the Bcl2 family induces Bax-dependent apoptosis

Increased expression of death receptors 4 and 5 synergizes the apoptosis response to combined treatment with etoposide and TRAIL

Control of mitochondrial membrane permeabilization by adenine nucleotide translocator interacting with HIV-1 viral protein rR and Bcl-2

Regulation of apoptosis by viral gene products

Respiratory syncytial virus infection sensitizes cells to apoptosis mediated by tumor necrosis factor-related apoptosis-inducing ligand

A novel influenza A virus mitochondrial protein that induces cell death.

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13132-13138

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10.1128/JVI.78.23.13132-13138.2004

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Kenneth L. Tyler

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2004-12-01T00:00:00Z

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15542665

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