Altered mineralization of human osteoarthritic osteoblasts is attributable to abnormal type I collagen production. - Wikidata - wikimedia - TriplyDB

Altered mineralization of human osteoarthritic osteoblasts is attributable to abnormal type I collagen production.

Altered mineralization of human osteoarthritic osteoblasts is attributable to abnormal type I collagen production.

http://www.wikidata.org/entity/Q37596567

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Transcriptional upregulation of DDR2 by ATF4 facilitates osteoblastic differentiation through p38 MAPK-mediated Runx2 activation On the Collagen Mineralization. A Review Osteoblast-chondrocyte interactions in osteoarthritis Bone-cartilage crosstalk: a conversation for understanding osteoarthritis Beneficial effect of resveratrol on phenotypic features and activity of osteoarthritic osteoblasts.Local leptin production in osteoarthritis subchondral osteoblasts may be responsible for their abnormal phenotypic expression.Gene expression analyses of subchondral bone in early experimental osteoarthritis by microarray.Osteoarthritis pathogenesis - a complex process that involves the entire joint.Generation of rodent and human osteoblasts.Regulation of osteoarthritis by omega-3 (n-3) polyunsaturated fatty acids in a naturally occurring model of disease.Potency Biomarker Signature Genes from Multiparametric Osteogenesis Assays: Will cGMP Human Bone Marrow Mesenchymal Stromal Cells Make Bone?Direct assessment of articular cartilage and underlying subchondral bone reveals a progressive gene expression change in human osteoarthritic knees.Basic science of osteoarthritis.Targeting subchondral bone for treating osteoarthritis: what is the evidence?Osteoblast physiology in normal and pathological conditions.The osteoarthritic niche and modulation of skeletal stem cell function for regenerative medicine.Bone remodelling in osteoarthritis.Effects of diacerein at the molecular level in the osteoarthritis disease process.PGE₂ and BMP-2 in bone and cartilage metabolism: 2 intertwining pathways.The coupling of bone and cartilage turnover in osteoarthritis: opportunities for bone antiresorptives and anabolics as potential treatments?Association of osteopontin with osteoarthritis.Alteration of Wnt5a expression and of the non-canonical Wnt/PCP and Wnt/PKC-Ca2+ pathways in human osteoarthritis osteoblasts.Impact of extracellular matrix derived from osteoarthritis subchondral bone osteoblasts on osteocytes: role of integrinβ1 and focal adhesion kinase signaling cues.What drives osteoarthritis?-synovial versus subchondral bone pathology.Osteoblast role in osteoarthritis pathogenesis.Role of melastatin transient receptor potential 7 channels in the osteoblastic differentiation of murine MC3T3 cells.GEORG-SCHMORL-PRIZE OF THE GERMAN SPINE SOCIETY (DWG) 2016: Comparison of in vitro osteogenic potential of iliac crest and degenerative facet joint bone autografts for intervertebral fusion in lumbar spinal stenosis.Interleukin-17 Can Induce Osteoarthritis in Rabbit Knee Joints Similar to Hulth's Method.New tools for studying osteoarthritis genetics in zebrafish.Ascorbic Acid Attenuates Senescence of Human Osteoarthritic Osteoblasts.Elevated Dickkopf-2 levels contribute to the abnormal phenotype of human osteoarthritic osteoblasts.Extracellular Vesicles from Adipose-Derived Mesenchymal Stem Cells Downregulate Senescence Features in Osteoarthritic Osteoblasts.Targeting subchondral bone mesenchymal stem cell activities for intrinsic joint repair in osteoarthritis.Alterations of Subchondral Bone Progenitor Cells in Human Knee and Hip Osteoarthritis Lead to a Bone Sclerosis Phenotype.Temporal changes in bone composition, architecture, and strength following estrogen deficiency in osteoporosis.R-spondins are newly recognized players in osteoarthritis that regulate Wnt signaling in osteoblasts.Elevated marrow inflammatory cells and osteoclasts in subchondral osteosclerosis in human knee osteoarthritis.In vivo bone-specific EphB4 overexpression in mice protects both subchondral bone and cartilage during osteoarthritis.Interleukin-6 from subchondral bone mesenchymal stem cells contributes to the pathological phenotypes of experimental osteoarthritis.Novel Ex Vivo Human Osteochondral Explant Model of Knee and Spine Osteoarthritis Enables Assessment of Inflammatory and Drug Treatment Responses.

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Altered mineralization of human osteoarthritic osteoblasts is attributable to abnormal type I collagen production.

http://www.wikidata.org/entity/Q37596567

description

article científic

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article scientifique

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bilimsel makale

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scientific article published on May 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Altered mineralization of huma ...... al type I collagen production.

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Altered mineralization of huma ...... al type I collagen production.

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type

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Altered mineralization of huma ...... al type I collagen production.

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Altered mineralization of huma ...... al type I collagen production.

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Altered mineralization of huma ...... al type I collagen production.

@en

Altered mineralization of huma ...... al type I collagen production.

@nl

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Arthritis & Rheumatology

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Altered mineralization of huma ...... al type I collagen production.

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Aline Delalandre

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Daniel Lajeunesse

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Denis Couchourel

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Isabelle Aubry

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Martin Lavigne

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P2860

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Measurement of protein using bicinchoninic acid

BMP-2-induced Runx2 expression is mediated by Dlx5, and TGF-beta 1 opposes the BMP-2-induced osteoblast differentiation by suppression of Dlx5 expression

Thermal stability and structure of cancellous bone mineral from the femoral head of patients with osteoarthritis or osteoporosis.

Mechanical and material properties of the subchondral bone plate from the femoral head of patients with osteoarthritis or osteoporosis

The electron microscope appearance of the subchondral bone plate in the human femoral head in osteoarthritis and osteoporosis.

In vivo bone-forming capacity of human bone marrow-derived stromal cells is stimulated by recombinant human bone morphogenetic protein-2.

Prediction of the progression of joint space narrowing in osteoarthritis of the knee by bone scintigraphy

How is Type I procollagen synthesis regulated at the gene level during tissue fibrosis.

Abnormal insulin-like growth factor 1 signaling in human osteoarthritic subchondral bone osteoblasts

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1438-1450

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scholarly article

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10.1002/ART.24489

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5

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60

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Jean-Pierre Pelletier

Johanne Martel-Pelletier

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2009-05-01T00:00:00Z

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24379695

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19404930

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