Sustained inflammasome activity in macrophages impairs wound healing in type 2 diabetic humans and mice. - Wikidata - wikimedia - TriplyDB

Sustained inflammasome activity in macrophages impairs wound healing in type 2 diabetic humans and mice.

Sustained inflammasome activity in macrophages impairs wound healing in type 2 diabetic humans and mice.

http://www.wikidata.org/entity/Q37597048

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The Innate Immune System in Acute and Chronic Wounds In vivo imaging reveals a pioneer wave of monocyte recruitment into mouse skin wounds Clinical challenges of chronic wounds: searching for an optimal animal model to recapitulate their complexity Notch Regulates Macrophage-Mediated Inflammation in Diabetic Wound Healing.Activation of poly(ADP-ribose) polymerase-1 delays wound healing by regulating keratinocyte migration and production of inflammatory mediators.Neuroprotectin/protectin D1: endogenous biosynthesis and actions on diabetic macrophages in promoting wound healing and innervation impaired by diabetes.Nlrp3-inflammasome activation in non-myeloid-derived cells aggravates diabetic nephropathy Contributions of cell subsets to cytokine production during normal and impaired wound healing.Nod-like receptor protein-3 inflammasome plays an important role during early stages of wound healing.Macrophage PPARγ and impaired wound healing in type 2 diabetes Oral Administration of Linoleic Acid Induces New Vessel Formation and Improves Skin Wound Healing in Diabetic Rats.Offloading treatment is linked to activation of proinflammatory cytokines and start of bone repair and remodeling in Charcot arthropathy patients.Diabetes medications: Impact on inflammation and wound healing Bisphosphonate Induces Osteonecrosis of the Jaw in Diabetic Mice via NLRP3/Caspase-1-Dependent IL-1β Mechanism.Melatonin promotes diabetic wound healing in vitro by regulating keratinocyte activity.NLRP3 Inflammasome Expression and Signaling in Human Diabetic Wounds and in High Glucose Induced Macrophages.The NLRP3 inflammasome in kidney disease and autoimmunity.Inflammasomes and dermatology.Drug delivery strategies to control macrophages for tissue repair and regeneration.Quercus infectoria inhibits Set7/NF-κB inflammatory pathway in macrophages exposed to a diabetic environment.Macrophage-based therapeutic strategies in regenerative medicine.Low-Magnitude High-Frequency Vibration Accelerated the Foot Wound Healing of n5-streptozotocin-induced Diabetic Rats by Enhancing Glucose Transporter 4 and Blood Microcirculation.Mitochondria-Targeted Antioxidant SkQ1 Improves Dermal Wound Healing in Genetically Diabetic Mice.Microbiota of Chronic Diabetic Wounds: Ecology, Impact, and Potential for Innovative Treatment Strategies.Deconvolution of heterogeneous wound tissue samples into relative macrophage phenotype composition via models based on gene expression.Immunomodulatory Effects of Human Cryopreserved Viable Amniotic Membrane in a Pro-Inflammatory Environment In Vitro.Drug delivery and tissue engineering to promote wound healing in the immunocompromised host: Current challenges and future directions.Heparan sulfate inhibits inflammation and improves wound healing by downregulating the NLR family pyrin domain containing 3 (NLRP3) inflammasome in diabetic rats.Activation of NLRP3 signalling accelerates skin wound healing.Changes in the extracellular matrix surrounding human chronic wounds revealed by 2-photon imaging.The Histone Methyltransferase MLL1 Directs Macrophage-Mediated Inflammation in Wound Healing and Is Altered in a Murine Model of Obesity and Type 2 Diabetes.Biomimetic Approaches for Bone Tissue Engineering.Distinct cytokine and chemokine patterns in chronic diabetic ulcers and acute wounds.Inhibition of NLRP3 Inflammasome Ameliorates Cerebral Ischemia-Reperfusion Injury in Diabetic Mice.Human αβ and γδ T Cells in Skin Immunity and Disease.Reactive oxygen species promote tubular injury in diabetic nephropathy: The role of the mitochondrial ros-txnip-nlrp3 biological axis.

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Sustained inflammasome activity in macrophages impairs wound healing in type 2 diabetic humans and mice.

http://www.wikidata.org/entity/Q37597048

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2013 nî lūn-bûn

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2013年學術文章

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Sustained inflammasome activit ...... pe 2 diabetic humans and mice.

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Sustained inflammasome activit ...... pe 2 diabetic humans and mice.

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Sustained inflammasome activit ...... pe 2 diabetic humans and mice.

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Eileen M Weinheimer-Haus

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Milie M Fang

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Rita E Mirza

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Timothy J Koh

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William J Ennis

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P2860

The serpin proteinase inhibitor 9 is an endogenous inhibitor of interleukin 1beta-converting enzyme (caspase-1) activity in human vascular smooth muscle cells

Caspase-1 (interleukin-1beta-converting enzyme) is inhibited by the human serpin analogue proteinase inhibitor 9

COPs and POPs: modulators of inflammasome activity

Non-canonical inflammasome activation targets caspase-11

Critical role for NALP3/CIAS1/Cryopyrin in innate and adaptive immunity through its regulation of caspase-1

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

The NLRP3 inflammasome instigates obesity-induced inflammation and insulin resistance

Inflammasomes in health and disease

The inflammasomes: guardians of the body

Cutaneous wound healing

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