A substance P antagonist inhibits vagally induced increase in vascular permeability and bronchial smooth muscle contraction in the guinea pig.
about
Interleukin-1ß-induced airway hyperresponsiveness enhances substance P in intrinsic neurons of ferret airwayAnatomy and neurophysiology of cough: CHEST Guideline and Expert Panel report.[D-Arg1,D-Phe5,D-Trp7,9,Leu11]substance P, a potent bombesin antagonist in murine Swiss 3T3 cells, inhibits the growth of human small cell lung cancer cells in vitro.Neutral endopeptidase and kininase II mediate glucocorticoid inhibition of neurogenic inflammation in the rat trachea.Peptidase modulation of the pulmonary effects of tachykinins in tracheal superfused guinea pig lungs.Maturation of neuromodulatory effect of substance P in rabbit airwaysHypertonic saline increases vascular permeability in the rat trachea by producing neurogenic inflammation.Neurogenic inflammation and asthma.Early Postnatal Ozone Exposure Alters Rat Nodose and Jugular Sensory Neuron Development.Neutral endopeptidase inhibitors potentiate substance P- and capsaicin-induced cough in awake guinea pigs.Sensory neuropeptide effects in human skin.Effects and interactions of sensory neuropeptides on airway microvascular leakage in guinea-pigs.Modulation of non-adrenergic, non-cholinergic neural bronchoconstriction in guinea-pig airways via GABAB-receptorsToluene diisocyanate increases airway responsiveness to substance P and decreases airway neutral endopeptidase.Recombinant human enkephalinase (neutral endopeptidase) prevents cough induced by tachykinins in awake guinea pigsMycoplasma pulmonis infections cause long-lasting potentiation of neurogenic inflammation in the respiratory tract of the ratOzone exposure initiates a sequential signaling cascade in airways involving interleukin-1beta release, nerve growth factor secretion, and substance P upregulationEffects of two novel tachykinin antagonists, FK224 and FK888, on neurogenic airway plasma exudation, bronchoconstriction and systemic hypotension in guinea-pigs in vivoProstacyclin activates tachykinin release from capsaicin-sensitive afferents in guinea-pig bronchi through a ruthenium red-sensitive pathway.The non-peptide tachykinin antagonist, CP-96,345, is a potent inhibitor of neurogenic inflammation.The involvement of sensory neuropeptides in toluene diisocyanate-induced tracheal hyperreactivity in the mouse airways.Suppression by neuropeptide Y of capsaicin-sensitive sensory nerve-mediated contraction in guinea-pig airways.Modulation of cholinergic neurotransmission in guinea-pig airways by opioids.Non-adrenergic, non-cholinergic contractions in the electrically field stimulated guinea-pig trachea.Substance P in sensory nerve fibres contributes to the development of oedema in the rat hind paw after thermal injury.Delay of cutaneous wound closure by morphine via local blockade of peripheral tachykinin release.The autonomic nervous system--a role in sudden infant death syndromePostjunctional inhibitory effect of the NK2 receptor antagonist, SR 48968, on sensory NANC bronchoconstriction in the guinea-pigEffects of two beta 3-adrenoceptor agonists, SR 58611A and BRL 37344, and of salbutamol on cholinergic and NANC neural contraction in guinea-pig main bronchi in vitro.Neuropeptides and asthma.Regulation of neurogenic inflammation by neutral endopeptidase.Interleukin (IL)-1 regulates ozone-enhanced tracheal smooth muscle responsiveness by increasing substance P (SP) production in intrinsic airway neurons of ferret.Prenatal and early, but not late, postnatal exposure of mice to sidestream tobacco smoke increases airway hyperresponsiveness later in life.High-affinity receptors for peptides of the bombesin family in Swiss 3T3 cells.CD10/neutral endopeptidase 24.11 hydrolyzes bombesin-like peptides and regulates the growth of small cell carcinomas of the lung.Tiotropium modulates transient receptor potential V1 (TRPV1) in airway sensory nerves: A beneficial off-target effect?Neuropeptide innervation of lymphoid organs.Neuropeptides and the pathogenesis of allergy.Substance P receptor-dependent responses of leukocytes in pulmonary inflammation.Dual roles of substance P: modulator of immune and neuroendocrine functions.
P2860
Q23924176-2381447A-5E1C-4797-885C-CFEABF7850DEQ27013738-D0E20B7E-AB16-4B4D-8756-71F7450AED96Q33560104-B91585CE-A854-412F-A0AF-E05B10A4D1E1Q34225017-61D45FAC-ED5E-4C0E-A24F-0DF6EB721BB4Q34240355-B1EC00D9-07F0-4144-ABA2-EDA2C559FA2BQ34241842-CEA7C91A-6E44-4417-AC39-7CA1A60C798DQ34253451-CB431099-D224-4E88-AACA-87AC4518FF50Q35256990-95DFA214-C3C4-487C-AD07-99B1BD36DE9AQ35580862-D9968036-A296-4A6C-8D46-D51871B1A61CQ35594884-DE2029B7-6C81-4128-BB49-A54BF73B8128Q35755140-FBF1F46D-D41A-4ABD-B73B-C2C5EFA36D2AQ35756064-CBF4B6DA-EB01-4221-8212-FCB39175A42DQ35756646-EE9066DB-C6E1-4B2F-BF1D-28702272BB1FQ35811207-D0C77439-AA1D-4026-9BD3-F6A255020470Q35813989-BE1BEA7C-4616-4FFA-A6E6-E46428349BB9Q35819000-B6A3829C-987D-4D2A-8CC1-79777EC6B3B8Q35819466-80EEA72F-1673-4C6B-AD9A-7AF40ACE377DQ35871462-35C81C07-33D0-4A2C-8F06-38AC0E20B409Q35871856-50A77B5B-4A10-4AD2-9A18-3A68DD8B6F13Q35872152-32D7DE30-31F5-4954-AD23-8E089DB810E2Q35884572-48952A5A-1E5C-4F7D-BFAE-165BBB1CA93DQ35887701-1CCB8ABE-B51A-490D-AFE9-1AEA870E541EQ35887903-DAC8F08E-0560-405A-A3C0-01380E8FFA3AQ35888520-545F6B8E-F8A2-4448-B6B0-EE8B5711F6A3Q36004414-F963D898-45BF-449E-B53A-41C438090065Q36019414-5ADB8682-F2C9-4022-A6AD-43529A0B4B51Q36181896-24EBF66A-C24F-421F-A9A9-7F488F15FFE4Q36329549-CEFD5CDD-43BA-4E7C-B815-FE70DC92DDB7Q36330020-83785B5B-6644-42B5-9BE7-6A3A4DBF8E19Q36633327-E5930BCE-C8B3-4213-9C08-A423A56178CEQ36755564-BC28F564-0950-43CB-806A-107C32FC1145Q37071480-0CEFE40C-B07D-4EF2-A478-46C7E0527893Q37331197-97A9F6EC-FC4B-44F4-8DB1-B1913B98FA22Q37554881-5CB6DAE2-32F6-41C8-9A72-9080BE41744BQ37632942-DBB824C4-BA70-4AAA-955B-B0C7EDBC34D1Q37672017-1F47517D-9699-474A-9D24-DBFCBA5DBE94Q37886967-2D8B5FAC-D693-4E5C-A204-51EE02E630EEQ38173434-D7E6B9B0-5C64-4D36-ADC4-FD0EE3D913FAQ38182073-0A224C05-ED95-49EE-9613-F935460A9FB3Q38186004-0884694A-B423-43C7-A0D5-20B332189224
P2860
A substance P antagonist inhibits vagally induced increase in vascular permeability and bronchial smooth muscle contraction in the guinea pig.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 1983
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
A substance P antagonist inhib ...... contraction in the guinea pig.
@en
A substance P antagonist inhib ...... contraction in the guinea pig.
@nl
type
label
A substance P antagonist inhib ...... contraction in the guinea pig.
@en
A substance P antagonist inhib ...... contraction in the guinea pig.
@nl
prefLabel
A substance P antagonist inhib ...... contraction in the guinea pig.
@en
A substance P antagonist inhib ...... contraction in the guinea pig.
@nl
P2093
P2860
P356
P1476
A substance P antagonist inhib ...... contraction in the guinea pig.
@en
P2093
P2860
P304
P356
10.1073/PNAS.80.4.1120
P407
P577
1983-02-01T00:00:00Z