Immune reconstitution: how it should work, what's broken, and why it matters. - Wikidata - wikimedia - TriplyDB

Immune reconstitution: how it should work, what's broken, and why it matters.

Immune reconstitution: how it should work, what's broken, and why it matters.

http://www.wikidata.org/entity/Q37612444

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Acute graft-versus-host disease: a bench-to-bedside update.Hematopoietic stem cell transplantation for multiple sclerosis: is it a clinical reality?Hematopoietic stem cell therapy for multiple sclerosis: top 10 lessons learned.Thymic recovery after allogeneic hematopoietic cell transplantation with non-myeloablative conditioning is limited to patients younger than 60 years of age Kinetics of IL-7 and IL-15 levels after allogeneic peripheral blood stem cell transplantation following nonmyeloablative conditioning Immune recovery in adult patients after myeloablative dual umbilical cord blood, matched sibling, and matched unrelated donor hematopoietic cell transplantation.Chimerism and tolerance without GVHD or engraftment syndrome in HLA-mismatched combined kidney and hematopoietic stem cell transplantation Feasibility and toxicity of hematopoietic stem cell transplant in multiple sclerosis.Expansion of a restricted residual host T reg-cell repertoire is dependent on IL-2 following experimental autologous hematopoietic stem transplantation.L-index as a novel index to evaluate both the intensity and duration of lymphopenia after allogeneic hematopoietic stem cell transplantation.

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Immune reconstitution: how it should work, what's broken, and why it matters.

http://www.wikidata.org/entity/Q37612444

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 October 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Immune reconstitution: how it should work, what's broken, and why it matters.

@en

Immune reconstitution: how it should work, what's broken, and why it matters.

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type

label

Immune reconstitution: how it should work, what's broken, and why it matters.

@en

Immune reconstitution: how it should work, what's broken, and why it matters.

@nl

prefLabel

Immune reconstitution: how it should work, what's broken, and why it matters.

@en

Immune reconstitution: how it should work, what's broken, and why it matters.

@nl

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J.BBMT.2009.10.003

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Biology of Blood and Marrow Transplantation

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Immune reconstitution: how it should work, what's broken, and why it matters.

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Ronald E Gress

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Stephen G Emerson

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William R Drobyski

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P2860

Age-dependent incidence, time course, and consequences of thymic renewal in adults

IL-7 administration to humans leads to expansion of CD8+ and CD4+ cells but a relative decrease of CD4+ T-regulatory cells

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Androgen receptors in thymic epithelium modulate thymus size and thymocyte development.

Assessment of thymic output in adults after haematopoietic stem-cell transplantation and prediction of T-cell reconstitution.

CCL25 increases thymopoiesis after androgen withdrawal

Maintaining the norm: T-cell homeostasis.

Sustained thymopoiesis and improvement in functional immunity induced by exogenous KGF administration in murine models of aging

Homeostatic expansion and repertoire regeneration of donor T cells during graft versus host disease is constrained by the host environment.

Absence of regulatory T-cell control of TH1 and TH17 cells is responsible for the autoimmune-mediated pathology in chronic graft-versus-host disease.

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scholarly article

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10.1016/J.BBMT.2009.10.003

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1 Suppl

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16

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2009-10-09T00:00:00Z

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