NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism. - Wikidata - wikimedia - TriplyDB

NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

http://www.wikidata.org/entity/Q37614231

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The transcription factors Ik-1 and MZF1 downregulate IGF-IR expression in NPM-ALK⁺ T-cell lymphoma microRNA-146a promotes mycobacterial survival in macrophages through suppressing nitric oxide production Leishmania (Leishmania) amazonensis induces macrophage miR-294 and miR-721 expression and modulates infection by targeting NOS2 and L-arginine metabolism.ALK-positive cancer: still a growing entity.Dysregulation of microRNAs and their association in the pathogenesis of T-cell lymphoma/leukemias.Crosstalk between microRNA and DNA Methylation Offers Potential Biomarkers and Targeted Therapies in ALK-Positive Lymphomas Concentration of circulating miRNA-containing particles in serum enhances miRNA detection and reflects CRC tissue-related deregulations.MiR-26a functions as a tumor suppressor in ambient particulate matter-bound metal-triggered lung cancer cell metastasis by targeting LIN28B-IL6-STAT3 axis.

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NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

http://www.wikidata.org/entity/Q37614231

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 14 March 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

@en

NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

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type

label

NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

@en

NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

@nl

prefLabel

NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

@en

NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.

@nl

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The Journal of Pathology

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NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism

@en

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Aarish Khan

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Deeksha Vishwamitra

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Haifeng Zhu

http://www.w3.org/2001/XMLSchema#string

Hesham M Amin

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Lixia Diao

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Roxsan Manshouri

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ALK, the chromosome 2 gene locus altered by the t(2;5) in non-Hodgkin's lymphoma, encodes a novel neural receptor tyrosine kinase that is highly related to leukocyte tyrosine kinase (LTK)

Nucleophosmin-anaplastic lymphoma kinase of large-cell anaplastic lymphoma is a constitutively active tyrosine kinase that utilizes phospholipase C-gamma to mediate its mitogenicity

Signal transducers and activators of transcription 3 (STAT3) inhibits transcription of the inducible nitric oxide synthase gene by interacting with nuclear factor kappaB

Characterization of the transforming activity of p80, a hyperphosphorylated protein in a Ki-1 lymphoma cell line with chromosomal translocation t(2;5)

miRNA-939 regulates human inducible nitric oxide synthase posttranscriptional gene expression in human hepatocytes

Widespread microRNA repression by Myc contributes to tumorigenesis

The PTEN-regulating microRNA miR-26a is amplified in high-grade glioma and facilitates gliomagenesis in vivo

Involvement of KSRP in the post-transcriptional regulation of human iNOS expression-complex interplay of KSRP with TTP and HuR.

Anaplastic lymphoma kinase (ALK) activates Stat3 and protects hematopoietic cells from cell death

Fusion of a kinase gene, ALK, to a nucleolar protein gene, NPM, in non-Hodgkin's lymphoma

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82-94

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scholarly article

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10.1002/PATH.4171

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1

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230

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Choladda V Curry

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2013-03-14T00:00:00Z

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234824356

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