G-CSF drives a posttraumatic immune program that protects the host from infection. - Wikidata - wikimedia - TriplyDB

G-CSF drives a posttraumatic immune program that protects the host from infection.

G-CSF drives a posttraumatic immune program that protects the host from infection.

http://www.wikidata.org/entity/Q37620301

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G-CSF drives a posttraumatic immune program that protects the host from infection.

http://www.wikidata.org/entity/Q37620301

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 27 January 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

G-CSF drives a posttraumatic immune program that protects the host from infection.

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G-CSF drives a posttraumatic immune program that protects the host from infection.

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type

label

G-CSF drives a posttraumatic immune program that protects the host from infection.

@en

G-CSF drives a posttraumatic immune program that protects the host from infection.

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prefLabel

G-CSF drives a posttraumatic immune program that protects the host from infection.

@en

G-CSF drives a posttraumatic immune program that protects the host from infection.

@nl

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JIMMUNOL.1302752

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Journal of Immunology

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G-CSF drives a posttraumatic immune program that protects the host from infection.

@en

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Cora K Ogle

http://www.w3.org/2001/XMLSchema#string

Francis X McCormack

http://www.w3.org/2001/XMLSchema#string

Jason C Gardner

http://www.w3.org/2001/XMLSchema#string

John G Noel

http://www.w3.org/2001/XMLSchema#string

Nikolaos M Nikolaidis

http://www.w3.org/2001/XMLSchema#string

Rebekah Karns

http://www.w3.org/2001/XMLSchema#string

P2860

Granulocyte-Colony Stimulating Factor (G-CSF) as an adjunct to antibiotics in the treatment of pneumonia in adults

IL-6 mediates hypoferremia of inflammation by inducing the synthesis of the iron regulatory hormone hepcidin

Myeloid-derived suppressor cells as regulators of the immune system

Bactericidal activity of testicular macrophages

Genomic responses in mouse models poorly mimic human inflammatory diseases

Divergent adaptive and innate immunological responses are observed in humans following blunt trauma.

STAT3 controls the neutrophil migratory response to CXCR2 ligands by direct activation of G-CSF-induced CXCR2 expression and via modulation of CXCR2 signal transduction

The use of flow cytometry to measure neutrophil function.

Hepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function.

Tumor-derived G-CSF facilitates neoplastic growth through a granulocytic myeloid-derived suppressor cell-dependent mechanism.

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2405-2417

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scholarly article

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10.4049/JIMMUNOL.1302752

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5

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2014-01-27T00:00:00Z

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259957300

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24470495

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3944070

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