Distinct roles of reactive nitrogen and oxygen species to control infection with the facultative intracellular bacterium Francisella tularensis.
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Differential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infectionsHigher Infection of Dengue Virus Serotype 2 in Human Monocytes of Patients with G6PD DeficiencyEffective, broad spectrum control of virulent bacterial infections using cationic DNA liposome complexes combined with bacterial antigensTularemia induces different biochemical responses in BALB/c mice and common volesEmrA1 membrane fusion protein of Francisella tularensis LVS is required for resistance to oxidative stress, intramacrophage survival and virulence in mice.Macrophage replication screen identifies a novel Francisella hydroperoxide resistance protein involved in virulence.Genome-wide RNAi screen in IFN-γ-treated human macrophages identifies genes mediating resistance to the intracellular pathogen Francisella tularensis.Francisella acid phosphatases inactivate the NADPH oxidase in human phagocytes.Low dose vaccination with attenuated Francisella tularensis strain SchuS4 mutants protects against tularemia independent of the route of vaccination.Glycogen synthase kinase-3beta (GSK3beta) inhibition suppresses the inflammatory response to Francisella infection and protects against tularemia in miceIKKβ in myeloid cells controls the host response to lethal and sublethal Francisella tularensis LVS infection.The Francisella tularensis LVS ΔpdpC mutant exhibits a unique phenotype during intracellular infectionInnate immune response to Anaplasma phagocytophilum contributes to hepatic injuryIron content differs between Francisella tularensis subspecies tularensis and subspecies holarctica strains and correlates to their susceptibility to H(2)O(2)-induced killingTryptophan prototrophy contributes to Francisella tularensis evasion of gamma interferon-mediated host defense.IglG and IglI of the Francisella pathogenicity island are important virulence determinants of Francisella tularensis LVSResistance of Francisella tularensis strains against reactive nitrogen and oxygen species with special reference to the role of KatG.Control of Francisella tularensis Intracellular Growth by Pulmonary Epithelial Cells.Inactivated Francisella tularensis live vaccine strain protects against respiratory tularemia by intranasal vaccination in an immunoglobulin A-dependent fashion.Francisella tularensis: taxonomy, genetics, and Immunopathogenesis of a potential agent of biowarfare.MglA regulates Francisella tularensis subsp. novicida (Francisella novicida) response to starvation and oxidative stressAlpha-1 antitrypsin is markedly decreased following pulmonary F. tularensis challengeInnate immunity against Francisella tularensis is dependent on the ASC/caspase-1 axis.Initial delay in the immune response to Francisella tularensis is followed by hypercytokinemia characteristic of severe sepsis and correlating with upregulation and release of damage-associated molecular patterns.IL-12Rβ2 is critical for survival of primary Francisella tularensis LVS infectionHeroin-induced conditioned immunomodulation requires expression of IL-1β in the dorsal hippocampus.Host immune response and acute disease in a zebrafish model of Francisella pathogenesisMAIT cells are critical for optimal mucosal immune responses during in vivo pulmonary bacterial infection.T cells from lungs and livers of Francisella tularensis-immune mice control the growth of intracellular bacteriaMyD88-dependent signaling prolongs survival and reduces bacterial burden during pulmonary infection with virulent Francisella tularensis.Contribution of citrulline ureidase to Francisella tularensis strain Schu S4 pathogenesisAn In Vitro Co-culture Mouse Model Demonstrates Efficient Vaccine-Mediated Control of Francisella tularensis SCHU S4 and Identifies Nitric Oxide as a Predictor of Efficacy.Loops and networks in control of Francisella tularensis virulence.Lack of OxyR and KatG Results in Extreme Susceptibility of Francisella tularensis LVS to Oxidative Stress and Marked Attenuation In vivo.Anti-CD25 antibody-mediated depletion of effector T cell populations enhances susceptibility of mice to acute but not chronic Toxoplasma gondii infection.T-bet regulates immunity to Francisella tularensis live vaccine strain infection, particularly in lungs.Immunity to Francisella.Basis for the failure of Francisella tularensis lipopolysaccharide to prime human polymorphonuclear leukocytes.Multifaceted effects of Francisella tularensis on human neutrophil function and lifespan.Control of intracellular Francisella tularensis by different cell types and the role of nitric oxide.
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Distinct roles of reactive nitrogen and oxygen species to control infection with the facultative intracellular bacterium Francisella tularensis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2004
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Distinct roles of reactive nit ...... terium Francisella tularensis.
@en
Distinct roles of reactive nit ...... terium Francisella tularensis.
@nl
type
label
Distinct roles of reactive nit ...... terium Francisella tularensis.
@en
Distinct roles of reactive nit ...... terium Francisella tularensis.
@nl
prefLabel
Distinct roles of reactive nit ...... terium Francisella tularensis.
@en
Distinct roles of reactive nit ...... terium Francisella tularensis.
@nl
P2093
P2860
P1476
Distinct roles of reactive nit ...... terium Francisella tularensis.
@en
P2093
Arne Tärnvik
Helena Lindgren
Stephan Stenmark
Wangxue Chen
P2860
P304
P356
10.1128/IAI.72.12.7172-7182.2004
P407
P577
2004-12-01T00:00:00Z