Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies. - Wikidata - wikimedia - TriplyDB

Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

http://www.wikidata.org/entity/Q37625670

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Lipoprotein(a) in cardiovascular diseases Thrombosis in ST-elevation myocardial infarction: Insights from thrombi retrieved by aspiration thrombectomy Monocyte Activation in Immunopathology: Cellular Test for Development of Diagnostics and Therapy Oxpholipin 11D: an anti-inflammatory peptide that binds cholesterol and oxidized phospholipids Angiotensin II enhances AT1-Nox1 binding and stimulates arterial smooth muscle cell migration and proliferation through AT1, Nox1, and interleukin-18.From hypertension to stroke: mechanisms and potential prevention strategies.Loss of Id3 increases VCAM-1 expression, macrophage accumulation, and atherogenesis in Ldlr-/- mice A pro-atherogenic HDL profile in coronary heart disease patients: an iTRAQ labelling-based proteomic approach Function and Therapeutic Potential of Mesenchymal Stem Cells in Atherosclerosis.The Therapeutic Potential of Anti-Inflammatory Exerkines in the Treatment of Atherosclerosis Spatial differences in the presence of FOXP3+ and GranzymeB+ T cells between the intra- and extravascular compartments in renal allograft vasculopathy.Association of MEF2A gene polymorphisms with coronary artery disease T cell-specific overexpression of TGFß1 fails to influence atherosclerosis in ApoE-deficient mice Myocardin regulates vascular smooth muscle cell inflammatory activation and disease Clonal restriction and predominance of regulatory T cells in coronary thrombi of patients with acute coronary syndromes.FoxO4 inhibits atherosclerosis through its function in bone marrow derived cells.High Incidence of ACE/PAI-1 in Association to a Spectrum of Other Polymorphic Cardiovascular Genes Involving PBMCs Proinflammatory Cytokines in Hypertensive Hypercholesterolemic Patients: Reversibility with a Combination of ACE Inhibitor and Statin.Atorvastatin Improves Inflammatory Response in Atherosclerosis by Upregulating the Expression of GARP.Delivery of Polymeric Nanoparticles to Target Vascular Diseases.Polymer-based therapeutics: nanoassemblies and nanoparticles for management of atherosclerosis Hyperglycemia and endothelial dysfunction in atherosclerosis: lessons from type 1 diabetes.A Unique Combination of Nutritionally Active Ingredients Can Prevent Several Key Processes Associated with Atherosclerosis In Vitro.Reliability of serum biomarkers of inflammation from repeated measures in healthy individuals Vaccination against T-cell epitopes of native ApoB100 reduces vascular inflammation and disease in a humanized mouse model of atherosclerosis.Depletion of FOXP3+ regulatory T cells promotes hypercholesterolemia and atherosclerosis Nanomedicines for Endothelial Disorders.Major vault protein regulates class A scavenger receptor-mediated tumor necrosis factor-α synthesis and apoptosis in macrophages.Omega-3 Fatty Acid Formulations in Cardiovascular Disease: Dietary Supplements are Not Substitutes for Prescription Products.The risk of atrial fibrillation in patients with gout: a nationwide population-based study.The comparative effects of atorvastatin and quince leaf extract on atherosclerosis Anti-inflammatory therapeutics for the treatment of atherosclerosis.Prognostic value of cystatin C in acute coronary syndromes: enhancer of atherosclerosis and promising therapeutic target.Role of dietary proteins and peptides in cardiovascular disease.Inflammation in atherosclerosis: a cause or a result of vascular disorders?The effects of extracellular matrix proteins on neutrophil-endothelial interaction--a roadway to multiple therapeutic opportunities.The immune system in stroke: clinical challenges and their translation to experimental research.Statins as modulators of regulatory T-cell biology.Design and utilization of macrophage and vascular smooth muscle cell co-culture systems in atherosclerotic cardiovascular disease investigation.Multiplex assay reliability and long-term intra-individual variation of serologic inflammatory biomarkers.Regulatory T cells in atherosclerosis: critical immune regulatory function and therapeutic potential.

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Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

http://www.wikidata.org/entity/Q37625670

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 30 October 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

@en

Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

@nl

type

label

Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

@en

Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

@nl

prefLabel

Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

@en

Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

@nl

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European Heart Journal

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Treating inflammation in atherosclerotic cardiovascular disease: emerging therapies.

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Göran K Hansson

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Roland Klingenberg

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P2860

Arachidonate 5-lipoxygenase promoter genotype, dietary arachidonic acid, and atherosclerosis

TNF/TNFR family members in costimulation of T cell responses

Global and regional burden of disease and risk factors, 2001: systematic analysis of population health data

MHC2TA is associated with differential MHC molecule expression and susceptibility to rheumatoid arthritis, multiple sclerosis and myocardial infarction

Inflammation, atherosclerosis, and coronary artery disease

Rosuvastatin to prevent vascular events in men and women with elevated C-reactive protein

Efficacy and safety of cholesterol-lowering treatment: prospective meta-analysis of data from 90,056 participants in 14 randomised trials of statins

Immunological and inflammatory functions of the interleukin-1 family

Lymphocyte recruitment into the aortic wall before and during development of atherosclerosis is partially L-selectin dependent.

An HMG-CoA reductase inhibitor, cerivastatin, suppresses growth of macrophages expressing matrix metalloproteinases and tissue factor in vivo and in vitro.

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2838-2844

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scholarly article

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10.1093/EURHEARTJ/EHP477

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23

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30

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2009-10-30T00:00:00Z

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38059086

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19880848

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atherosclerosis

cardiovascular disease