PI3K/AKT signaling is essential for communication between tissue-infiltrating mast cells, macrophages, and epithelial cells in colitis-induced cancer.
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β-Catenin promotes colitis and colon cancer through imprinting of proinflammatory properties in T cells.A strategy to identify dominant point mutant modifiers of a quantitative trait.Immunoglobulin free light chains are biomarkers of poor prognosis in basal-like breast cancer and are potential targets in tumor-associated inflammationRecent advancement in understanding colitis-associated tumorigenesis.Lentiviral shRNA against KCa3.1 inhibits allergic response in allergic rhinitis and suppresses mast cell activity via PI3K/AKT signaling pathway.Loss of PHLPP protects against colitis by inhibiting intestinal epithelial cell apoptosis.Network modelling reveals the mechanism underlying colitis-associated colon cancer and identifies novel combinatorial anti-cancer targets.Cross-talk between intestinal epithelial cells and immune cells in inflammatory bowel disease.Adipose tissue insulin sensitivity and macrophage recruitment: Does PI3K pick the pathway?Novel methylxanthine derivative-mediated anti-inflammatory effects in inflammatory bowel disease.Reactive Oxygen Species Evoked by Potassium Deprivation and Staurosporine Inactivate Akt and Induce the Expression of TXNIP in Cerebellar Granule Neurons.The Role of Mast Cells in Molding the Tumor Microenvironment.MFG-E8 overexpression promotes colorectal cancer progression via AKT/MMPs signalling.Activation of protease-activated receptor 2 reduces glioblastoma cell apoptosis.Redirecting tumor-associated macrophages to become tumoricidal effectors as a novel strategy for cancer therapy.Mast cells positive to tryptase and tumour-associated macrophages correlate with angiogenesis in locally advanced colorectal cancer patients undergone to surgery.Aloperine Protects Mice against DSS-Induced Colitis by PP2A-Mediated PI3K/Akt/mTOR Signaling Suppression.The STAT3 inhibitor pyrimethamine displays anti-cancer and immune stimulatory effects in murine models of breast cancer.Human mast cells promote colon cancer growth via bidirectional crosstalk: studies in 2D and 3D coculture models
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PI3K/AKT signaling is essential for communication between tissue-infiltrating mast cells, macrophages, and epithelial cells in colitis-induced cancer.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 13 March 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
PI3K/AKT signaling is essentia ...... lls in colitis-induced cancer.
@en
PI3K/AKT signaling is essentia ...... lls in colitis-induced cancer.
@nl
type
label
PI3K/AKT signaling is essentia ...... lls in colitis-induced cancer.
@en
PI3K/AKT signaling is essentia ...... lls in colitis-induced cancer.
@nl
prefLabel
PI3K/AKT signaling is essentia ...... lls in colitis-induced cancer.
@en
PI3K/AKT signaling is essentia ...... lls in colitis-induced cancer.
@nl
P2093
P2860
P1476
PI3K/AKT signaling is essentia ...... lls in colitis-induced cancer.
@en
P2093
Ali Keshavarzian
David J Bentrem
Eric C Cheon
Fu-Nien Tsai
Joshua E Melson
Khashayarsha Khazaie
Mohammad W Khan
Nichole R Blatner
Philipp Beckhove
P2860
P304
P356
10.1158/1078-0432.CCR-12-2623
P407
P577
2013-03-13T00:00:00Z