A PTBA small molecule enhances recovery and reduces postinjury fibrosis after aristolochic acid-induced kidney injury. - Wikidata - wikimedia - TriplyDB

A PTBA small molecule enhances recovery and reduces postinjury fibrosis after aristolochic acid-induced kidney injury.

A PTBA small molecule enhances recovery and reduces postinjury fibrosis after aristolochic acid-induced kidney injury.

http://www.wikidata.org/entity/Q37629173

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Epigenetics in acute kidney injury Progression after AKI: Understanding Maladaptive Repair Processes to Predict and Identify Therapeutic Treatments.Zebrafish Models of Kidney Damage and Repair Class I HDAC activity is required for renal protection and regeneration after acute kidney injury Treatment of chronic kidney diseases with histone deacetylase inhibitors.The nucleosome remodeling and deacetylase complex in development and disease.Delayed treatment with PTBA analogs reduces postinjury renal fibrosis after kidney injury.Little fish, big catch: zebrafish as a model for kidney disease Bioinformatics facilitating the use of microarrays to delineate potential miRNA biomarkers in aristolochic acid nephropathy.Animal models of regression/progression of kidney disease Epigenetic regulation in the acute kidney injury (AKI) to chronic kidney disease transition (CKD).Noncoding RNA and epigenetic gene regulation in renal diseases.Pyridoxamine reduces postinjury fibrosis and improves functional recovery after acute kidney injury Translating Knowledge Into Therapy for Acute Kidney Injury.Endocycle-related tubular cell hypertrophy and progenitor proliferation recover renal function after acute kidney injury.

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A PTBA small molecule enhances recovery and reduces postinjury fibrosis after aristolochic acid-induced kidney injury.

http://www.wikidata.org/entity/Q37629173

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 26 December 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

A PTBA small molecule enhances ...... ic acid-induced kidney injury.

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A PTBA small molecule enhances ...... ic acid-induced kidney injury.

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A PTBA small molecule enhances ...... ic acid-induced kidney injury.

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A PTBA small molecule enhances ...... ic acid-induced kidney injury.

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A PTBA small molecule enhances ...... ic acid-induced kidney injury.

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A PTBA small molecule enhances ...... ic acid-induced kidney injury.

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AJPRENAL.00534.2013

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American Journal of Physiology - Renal Physiology

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A PTBA small molecule enhances ...... ic acid-induced kidney injury.

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Ke Xin Zhang

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Lee McDermott

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Mark P de Caestecker

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Neil A Hukriede

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Paisit Paueksakon

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Takuto Chiba

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Tatiana Novitskaya

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P2860

Mitotic phosphorylation of histone H3: spatio-temporal regulation by mammalian Aurora kinases

Histone deacetylase inhibition reduces myocardial ischemia-reperfusion injury in mice

The renal mononuclear phagocytic system

F4/80, a monoclonal antibody directed specifically against the mouse macrophage

Macrophage Wnt7b is critical for kidney repair and regeneration

Macrophage polarization: tumor-associated macrophages as a paradigm for polarized M2 mononuclear phagocytes

The chemokine receptors CCR2 and CX3CR1 mediate monocyte/macrophage trafficking in kidney ischemia-reperfusion injury

Pressure activates epidermal growth factor receptor leading to the induction of iNOS via NFkappaB and STAT3 in human proximal tubule cells.

Activation of p53 promotes renal injury in acute aristolochic acid nephropathy.

Urinary chemokine (C-C motif) ligand 2 (monocyte chemotactic protein-1) as a tubular injury marker for early detection of cisplatin-induced nephrotoxicity.

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F496-504

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10.1152/AJPRENAL.00534.2013

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2013-12-26T00:00:00Z

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24370591

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