Suppression of PPARγ through MKRN1-mediated ubiquitination and degradation prevents adipocyte differentiation.
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Cell Models and Their Application for Studying Adipogenic Differentiation in Relation to Obesity: A ReviewPPARs: Protectors or Opponents of Myocardial Function?A new pathway in the control of the initiation of puberty: the MKRN3 genePutative regulatory factors associated with intramuscular fat contentAblation of galectin-3 induces p27(KIP1)-dependent premature senescence without oncogenic stressUbiquitylation of nuclear receptors: new linkages and therapeutic implications.Analysis of RNA Interference Lines Identifies New Functions of Maternally-Expressed Genes Involved in Embryonic Patterning in Drosophila melanogaster.Modulation of host ubiquitin system genes in human endometrial cell line infected with Mycobacterium tuberculosis.MuRF2 regulates PPARγ1 activity to protect against diabetic cardiomyopathy and enhance weight gain induced by a high fat diet.Makorin ortholog LEP-2 regulates LIN-28 stability to promote the juvenile-to-adult transition in Caenorhabditis elegans.F-box only protein 9 is an E3 ubiquitin ligase of PPARγ.Ubiquitin Ligase NEDD4 Regulates PPARγ Stability and Adipocyte Differentiation in 3T3-L1 Cells.C-terminus of HSC70-Interacting Protein (CHIP) Inhibits Adipocyte Differentiation via Ubiquitin- and Proteasome-Mediated Degradation of PPARγ.The multifaceted factor peroxisome proliferator-activated receptor γ (PPARγ) in metabolism, immunity, and cancer.Activation of nuclear PTEN by inhibition of Notch signaling induces G2/M cell cycle arrest in gastric cancer.Tetrahydrocannabinolic acid is a potent PPARγ agonist with neuroprotective activity.Genome-wide association study of body mass index in subjects with alcohol dependence.Human adenovirus infection causes the cellular MKRN1 E3 ubiquitin ligase degradation involving the viral core protein pVII.Identification of differentially expressed genes and pathways for intramuscular fat metabolism between breast and thigh tissues of chickens.Hepatic PPARα function is controlled by polyubiquitination and proteasome-mediated degradation via the coordinated actions of PAQR3 and HUWE1.Grail is involved in adipocyte differentiation and diet-induced obesity.Functional Regulation of PPARs through Post-Translational Modifications.Loss of the E3 ubiquitin ligase MKRN1 represses diet-induced metabolic syndrome through AMPK activation
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Suppression of PPARγ through MKRN1-mediated ubiquitination and degradation prevents adipocyte differentiation.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 13 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Suppression of PPARγ through M ...... nts adipocyte differentiation.
@en
Suppression of PPARγ through M ...... nts adipocyte differentiation.
@nl
type
label
Suppression of PPARγ through M ...... nts adipocyte differentiation.
@en
Suppression of PPARγ through M ...... nts adipocyte differentiation.
@nl
prefLabel
Suppression of PPARγ through M ...... nts adipocyte differentiation.
@en
Suppression of PPARγ through M ...... nts adipocyte differentiation.
@nl
P2093
P2860
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P1476
Suppression of PPARγ through M ...... nts adipocyte differentiation.
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P2093
P2860
P2888
P304
P356
10.1038/CDD.2013.181
P50
P577
2013-12-13T00:00:00Z
P6179
1029546874