Epigenetic Regulation of Axon Regeneration after Neural Injury. - Wikidata - wikimedia - TriplyDB

Epigenetic Regulation of Axon Regeneration after Neural Injury.

Epigenetic Regulation of Axon Regeneration after Neural Injury.

http://www.wikidata.org/entity/Q37637863

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Deep Sequencing Reveals the Significant Involvement of cAMP-Related Signaling Pathways Following Sciatic Nerve Crush.Neuromuscular degenerative effects of Ankaferd Blood Stopper® in mouse sciatic nerve model.

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Epigenetic Regulation of Axon Regeneration after Neural Injury.

http://www.wikidata.org/entity/Q37637863

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 26 January 2017

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Epigenetic Regulation of Axon Regeneration after Neural Injury.

@en

Epigenetic Regulation of Axon Regeneration after Neural Injury.

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type

label

Epigenetic Regulation of Axon Regeneration after Neural Injury.

@en

Epigenetic Regulation of Axon Regeneration after Neural Injury.

@nl

prefLabel

Epigenetic Regulation of Axon Regeneration after Neural Injury.

@en

Epigenetic Regulation of Axon Regeneration after Neural Injury.

@nl

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MOLCELLS.2017.2311

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Molecules and Cells

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Epigenetic Regulation of Axon Regeneration after Neural Injury.

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Yongcheol Cho

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Promoting axon regeneration in the adult CNS by modulation of the PTEN/mTOR pathway

Glial inhibition of CNS axon regeneration

What makes a RAG regeneration associated?

HDAC signaling in neuronal development and axon regeneration

Nogo-A is a myelin-associated neurite outgrowth inhibitor and an antigen for monoclonal antibody IN-1

Oligodendrocyte-myelin glycoprotein is a Nogo receptor ligand that inhibits neurite outgrowth

Role of axonal transport in neurodegenerative diseases

The transcriptional landscape of dorsal root ganglia after sciatic nerve transection

HDAC inhibition promotes neuronal outgrowth and counteracts growth cone collapse through CBP/p300 and P/CAF-dependent p53 acetylation

HDAC6 is a target for protection and regeneration following injury in the nervous system

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scholarly article

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10.14348/MOLCELLS.2017.2311

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