Bystander suppression of allergic airway inflammation by lung resident memory CD8+ T cells.
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Interactions between commensal intestinal bacteria and the immune systemMacaque proteome response to highly pathogenic avian influenza and 1918 reassortant influenza virus infectionsCytotoxic potential of lung CD8(+) T cells increases with chronic obstructive pulmonary disease severity and with in vitro stimulation by IL-18 or IL-15.Influenza infection in suckling mice expands an NKT cell subset that protects against airway hyperreactivity.The increased prevalence of allergy and the hygiene hypothesis: missing immune deviation, reduced immune suppression, or both?CD8+ T Cells Mediate Female-Dominant IL-4 Production and Airway Inflammation in Allergic Asthma.The sickle cell mouse lung: proinflammatory and primed for allergic inflammation.Innate responsiveness of CD8 memory T-cell populations nonspecifically inhibits allergic sensitization.Respiratory virus-induced regulation of asthma-like responses in mice depends upon CD8 T cells and interferon-gamma production.T-bet negatively regulates autoimmune myocarditis by suppressing local production of interleukin 17T cell memory in the lung airways.The immunopathogenesis of chronic obstructive pulmonary disease: insights from recent research.Pathogens and immunologic memory in asthma: what have we learned?The chemokine receptor CCR5 plays a key role in the early memory CD8+ T cell response to respiratory virus infections.Divergent effects of biolistic gene transfer in a mouse model of allergic airway inflammation.Unravelling synergistic immune interactions between respiratory virus infections and allergic airway inflammation.Induction of T helper type 1-like regulatory cells that express Foxp3 and protect against airway hyper-reactivity.Sialyl lewisx antigen-expressing human CD4+ T and CD8+ T cells as initial immune responders in memory phenotype subsets.Secretory products from infective forms of Nippostrongylus brasiliensis induce a rapid allergic airway inflammatory response.Allergic airway inflammation is exacerbated during acute influenza infection and correlates with increased allergen presentation and recruitment of allergen-specific T-helper type 2 cells.Antigen-specific cytotoxic T lymphocytes target airway CD103+ and CD11b+ dendritic cells to suppress allergic inflammation.Critical role for syndecan-4 in dendritic cell migration during development of allergic airway inflammation.Intranasal delivery of whole influenza vaccine prevents subsequent allergen-induced sensitization and airway hyper-reactivity in mice.
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P2860
Bystander suppression of allergic airway inflammation by lung resident memory CD8+ T cells.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 12 April 2004
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Bystander suppression of aller ...... resident memory CD8+ T cells.
@en
Bystander suppression of aller ...... resident memory CD8+ T cells.
@nl
type
label
Bystander suppression of aller ...... resident memory CD8+ T cells.
@en
Bystander suppression of aller ...... resident memory CD8+ T cells.
@nl
prefLabel
Bystander suppression of aller ...... resident memory CD8+ T cells.
@en
Bystander suppression of aller ...... resident memory CD8+ T cells.
@nl
P2093
P2860
P50
P356
P1476
Bystander suppression of aller ...... resident memory CD8+ T cells.
@en
P2093
Benjamin J Marsland
Mali Camberis
Sarah M Hook
P2860
P304
P356
10.1073/PNAS.0401582101
P407
P577
2004-04-12T00:00:00Z