Inactivation of AKT induces cellular senescence in uterine leiomyoma. - Wikidata - wikimedia - TriplyDB

Inactivation of AKT induces cellular senescence in uterine leiomyoma.

Inactivation of AKT induces cellular senescence in uterine leiomyoma.

http://www.wikidata.org/entity/Q37647999

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Therapeutic targeting of replicative immortality Role of miR-182 in response to oxidative stress in the cell fate of human fallopian tube epithelial cells.Potential Diagnostic and Prognostic Value of Plasma Circulating MicroRNA-182 in Human Glioma.Dysfunctional MnSOD leads to redox dysregulation and activation of prosurvival AKT signaling in uterine leiomyomas.Asxl1 deficiency in embryonic fibroblasts leads to cellular senescence via impairment of the AKT-E2F pathway and Ezh2 inactivation.Targeting leiomyomas with all-trans-retinoic acid at phosphoinositide 3-kinase pathway suppression: Effective roles of β-catenin and of signaling interactions.Circulating microRNA-182 in plasma and its potential diagnostic and prognostic value for pancreatic cancer.Immunoexpression of p16 in uterine leiomyomas with infarct-type necrosis: an analysis of 35 cases.The AKT/BCL-2 axis mediates survival of uterine leiomyoma in a novel 3D spheroid model.

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Inactivation of AKT induces cellular senescence in uterine leiomyoma.

http://www.wikidata.org/entity/Q37647999

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 29 January 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Inactivation of AKT induces cellular senescence in uterine leiomyoma.

@en

Inactivation of AKT induces cellular senescence in uterine leiomyoma.

@nl

type

label

Inactivation of AKT induces cellular senescence in uterine leiomyoma.

@en

Inactivation of AKT induces cellular senescence in uterine leiomyoma.

@nl

prefLabel

Inactivation of AKT induces cellular senescence in uterine leiomyoma.

@en

Inactivation of AKT induces cellular senescence in uterine leiomyoma.

@nl

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Inactivation of AKT induces cellular senescence in uterine leiomyoma.

@en

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Beihua Kong

http://www.w3.org/2001/XMLSchema#string

J Julie Kim

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Jian-Jun Wei

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Vania Vidimar

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Wenan Qiang

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Xiaofei Xu

http://www.w3.org/2001/XMLSchema#string

Zhenxiao Lu

http://www.w3.org/2001/XMLSchema#string

P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

A novel role for high-mobility group a proteins in cellular senescence and heterochromatin formation

Protein kinases and phosphatases in the control of cell fate

First-in-man clinical trial of the oral pan-AKT inhibitor MK-2206 in patients with advanced solid tumors.

Antiproliferative effects by Let-7 repression of high-mobility group A2 in uterine leiomyoma

Combination treatment with MEK and AKT inhibitors is more effective than each drug alone in human non-small cell lung cancer in vitro and in vivo

Cellular survival: a play in three Akts

Modulation of microRNA processing by p53

Cellular senescence in usual type uterine leiomyoma.

Profiling and functional analyses of microRNAs and their target gene products in human uterine leiomyomas.

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1510-1519

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scholarly article

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10.1210/EN.2013-1929

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