Monoamine oxidase is a major determinant of redox balance in human atrial myocardium and is associated with postoperative atrial fibrillation. - Wikidata - wikimedia - TriplyDB

Monoamine oxidase is a major determinant of redox balance in human atrial myocardium and is associated with postoperative atrial fibrillation.

Monoamine oxidase is a major determinant of redox balance in human atrial myocardium and is associated with postoperative atrial fibrillation.

http://www.wikidata.org/entity/Q37648518

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The Role of Mitochondrial Reactive Oxygen Species in Cardiovascular Injury and Protective Strategies Mitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioning From ATP to PTP and Back: A Dual Function for the Mitochondrial ATP Synthase Reactive Oxygen-Related Diseases: Therapeutic Targets and Emerging Clinical Indications Monoamine Oxidases as Potential Contributors to Oxidative Stress in Diabetes: Time for a Study in Patients Undergoing Heart Surgery Monoamine Oxidases, Oxidative Stress, and Altered Mitochondrial Dynamics in Cardiac Ageing Reactive oxygen species and redox compartmentalization Monoamine oxidase inhibition prevents mitochondrial dysfunction and apoptosis in myoblasts from patients with collagen VI myopathies.Obesity in a model of gpx4 haploinsufficiency uncovers a causal role for lipid-derived aldehydes in human metabolic disease and cardiomyopathy.Selective downregulation of mitochondrial electron transport chain activity and increased oxidative stress in human atrial fibrillation.Monoamine Oxidase Is Overactivated in Left and Right Ventricles from Ischemic Hearts: An Intriguing Therapeutic Target.The "Goldilocks Zone" from a redox perspective-Adaptive vs. deleterious responses to oxidative stress in striated muscle.β-Adrenergic signaling, monoamine oxidase A and antioxidant defence in the myocardium of SHR and SHR-mtBN conplastic rat strains: the effect of chronic hypoxia.Basic Biology of Oxidative Stress and the Cardiovascular System: Part 1 of a 3-Part Series.Assessment of Mitochondrial Dysfunction and Monoamine Oxidase Contribution to Oxidative Stress in Human Diabetic Hearts.Mitochondrial oxidative stress promotes atrial fibrillation.Defining the role of oxidative stress in atrial fibrillation and diabetes.Hyperthyroidism, but not hypertension, impairs PITX2 expression leading to Wnt-microRNA-ion channel remodeling.Plasma Catecholamine Levels on the Morning of Surgery Predict Post-Operative Atrial Fibrillation.Oleuropein Aglycone Protects against MAO-A-Induced Autophagy Impairment and Cardiomyocyte Death through Activation of TFEB.Calcium in the Pathophysiology of Atrial Fibrillation and Heart Failure

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Monoamine oxidase is a major determinant of redox balance in human atrial myocardium and is associated with postoperative atrial fibrillation.

http://www.wikidata.org/entity/Q37648518

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 26 February 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Monoamine oxidase is a major d ...... operative atrial fibrillation.

@en

Monoamine oxidase is a major d ...... operative atrial fibrillation.

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type

label

Monoamine oxidase is a major d ...... operative atrial fibrillation.

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Monoamine oxidase is a major d ...... operative atrial fibrillation.

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prefLabel

Monoamine oxidase is a major d ...... operative atrial fibrillation.

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Monoamine oxidase is a major d ...... operative atrial fibrillation.

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P356

JAHA.113.000713

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Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease

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Monoamine oxidase is a major d ...... toperative atrial fibrillation

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Alan P Kypson

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Curtis A Anderson

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Ethan J Anderson

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Evelio Rodriguez

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J Mark Williams

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Jimmy T Efird

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Kathleen A Thayne

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Lalage A Katunga

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Linda C Kindell

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Stephen W Davies

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P2860

Monoamine oxidase B prompts mitochondrial and cardiac dysfunction in pressure overloaded hearts

Increased propensity for cell death in diabetic human heart is mediated by mitochondrial-dependent pathways

Molecular and mechanistic properties of the membrane-bound mitochondrial monoamine oxidases

Redox environment of the cell as viewed through the redox state of the glutathione disulfide/glutathione couple

The prevention and treatment of missing data in clinical trials

Allopurinol enhances the contractile response to dobutamine and exercise in dogs with pacing-induced heart failure.

Substrate-specific derangements in mitochondrial metabolism and redox balance in the atrium of the type 2 diabetic human heart.

Monoamine oxidase A-mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload.

The role of neuronal and extraneuronal plasma membrane transporters in the inactivation of peripheral catecholamines.

Inflammation as a risk factor for atrial fibrillation.

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e000713

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10.1161/JAHA.113.000713

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Wesley T O'Neal

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2014-02-26T00:00:00Z

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24572256

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atrial fibrillation

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