Challenges and opportunities identifying therapeutic targets for chemotherapy-induced peripheral neuropathy resulting from oxidative DNA damage.
about
DNA damage mediates changes in neuronal sensitivity induced by the inflammatory mediators, MCP-1 and LPS, and can be reversed by enhancing the DNA repair function of APE1.APE1/Ref-1 redox-specific inhibition decreases survivin protein levels and induces cell cycle arrest in prostate cancer cells.Exploiting the Ref-1-APE1 node in cancer signaling and other diseases: from bench to clinic.APX3330 Promotes Neurorestorative Effects after Stroke in Type One Diabetic Rats.Blocking HIF signaling via novel inhibitors of CA9 and APE1/Ref-1 dramatically affects pancreatic cancer cell survival
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Challenges and opportunities identifying therapeutic targets for chemotherapy-induced peripheral neuropathy resulting from oxidative DNA damage.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on January 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
@da
vědecký článek
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name
Challenges and opportunities i ...... ing from oxidative DNA damage.
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Challenges and opportunities i ...... ing from oxidative DNA damage.
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type
label
Challenges and opportunities i ...... ing from oxidative DNA damage.
@en
Challenges and opportunities i ...... ing from oxidative DNA damage.
@nl
prefLabel
Challenges and opportunities i ...... ing from oxidative DNA damage.
@en
Challenges and opportunities i ...... ing from oxidative DNA damage.
@nl
P2860
P356
P1476
Challenges and opportunities i ...... ting from oxidative DNA damage
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P2860
P356
10.4103/1673-5374.198986
P407
P577
2017-01-01T00:00:00Z