Interferon response factor 3 is crucial to poly-I:C induced NK cell activity and control of B16 melanoma growth.
about
TLR-3 stimulation improves anti-tumor immunity elicited by dendritic cell exosome-based vaccines in a murine model of melanoma.The cryo-thermal therapy eradicated melanoma in mice by eliciting CD4+ T-cell-mediated antitumor memory immune response.Significant role for IRF3 in both T cell and APC effector functions during T cell responses.Hepatitis A virus cellular receptor 2 (HAVCR2) is decreased with viral infection and regulates pro-labour mediators OA.Activation of IRF3 contributes to IFN-γ and ISG54 expression during the immune responses to B16F10 tumor growth.
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Interferon response factor 3 is crucial to poly-I:C induced NK cell activity and control of B16 melanoma growth.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 22 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Interferon response factor 3 i ...... ontrol of B16 melanoma growth.
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Interferon response factor 3 i ...... ontrol of B16 melanoma growth.
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type
label
Interferon response factor 3 i ...... ontrol of B16 melanoma growth.
@en
Interferon response factor 3 i ...... ontrol of B16 melanoma growth.
@nl
prefLabel
Interferon response factor 3 i ...... ontrol of B16 melanoma growth.
@en
Interferon response factor 3 i ...... ontrol of B16 melanoma growth.
@nl
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Interferon response factor 3 i ...... ontrol of B16 melanoma growth.
@en
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Deborah M Brown
Phyllis M Kumm
Thomas M Petro
Tyler C Moore
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10.1016/J.CANLET.2013.12.022
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P577
2013-12-22T00:00:00Z